Intrinsic-mediated caspase activation is essential for cardiomyocyte hypertrophy (Q24605583)

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27 February 2020

title

Intrinsic-mediated caspase activation is essential for cardiomyocyte hypertrophy (English)

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27 February 2020

7

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27 February 2020

author name string

Charis Putinski

1

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27 February 2020

Mohammad Abdul-Ghani

2

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27 February 2020

Rebecca Stiles

3

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27 February 2020

Steve Brunette

4

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27 February 2020

Sarah A Dick

5

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27 February 2020

Pasan Fernando

6

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27 February 2020

language of work or name

English

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publication date

7 October 2013

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Proceedings of the National Academy of Sciences of the United States of America

27 February 2020

published in

1 reference

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27 February 2020

volume

110

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27 February 2020

issue

43

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27 February 2020

page(s)

E4079-87

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describes a project that uses

27 February 2020

ImageJ

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11 June 2022

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inferred from PubMed Central ID database lookup

cites work

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Metacaspase Yca1 is required for clearance of insoluble protein aggregates.

7 April 2017

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Functional consequences of caspase activation in cardiac myocytes

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MEF2 is upregulated during cardiac hypertrophy and is required for normal post-natal growth of the myocardium

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Interaction between NFκB and NFAT coordinates cardiac hypertrophy and pathological remodeling

27 September 2017

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Endonuclease G is a novel determinant of cardiac hypertrophy and mitochondrial function

27 September 2017

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Wnt11 promotes cardiomyocyte development by caspase-mediated suppression of canonical Wnt signals

27 September 2017

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Having a change of heart: reversing the suicidal proclivities of cardiac myocytes.

27 September 2017

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TNF-alpha inhibition attenuates adverse myocardial remodeling in a rat model of volume overload

27 September 2017

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Noxa: at the tip of the balance between life and death.

27 September 2017

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Molecular mechanisms underlying the transition of cardiac hypertrophy to heart failure

27 September 2017

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27 September 2017

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27 September 2017

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27 September 2017

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27 September 2017

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27 September 2017

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27 September 2017

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27 September 2017

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27 September 2017

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27 September 2017

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27 September 2017

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27 September 2017

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27 September 2017

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27 September 2017

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27 September 2017

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A dynamic and chamber-specific mitochondrial remodeling in right ventricular hypertrophy can be therapeutically targeted

30 May 2018

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Cleavage and cytoplasmic relocalization of histone deacetylase 3 are important for apoptosis progression

21 January 2018

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Maintenance of cardiac energy metabolism by histone deacetylase 3 in mice

21 January 2018

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An evolutionary conserved pathway of nuclear factor-kappaB activation involving caspase-mediated cleavage and N-end rule pathway-mediated degradation of IkappaBalpha

21 January 2018

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Cardiomyocyte apoptosis induced by Galphaq signaling is mediated by permeability transition pore formation and activation of the mitochondrial death pathway.

21 January 2018

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Caspase-3 inhibition preserves myocardial geometry and long-term function after infarction.

21 January 2018

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Determination of the matrix free Ca2+ concentration and kinetics of Ca2+ efflux in liver and heart mitochondria

21 January 2018

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12 December 2020

Emerging role of Mcl-1 in actively counteracting BH3-only proteins in apoptosis

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12 December 2020

The possible role of caspase-3 in pathological and physiological cardiac hypertrophy in rats

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12 December 2020

Tumor necrosis factor-alpha mediates cardiac remodeling and ventricular dysfunction after pressure overload state

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12 December 2020

Identifiers

DOI

10.1073/PNAS.1315587110

2 references

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:24101493%20AND%20SRC:MED&resulttype=core&format=json

27 February 2020

ADS bibcode

2013PNAS..110E4079P

0 references

1 reference

2 references

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:24101493%20AND%20SRC:MED&resulttype=core&format=json

27 February 2020

PubMed publication ID

24101493

2 references