Epstein-Barr virus-induced miR-155 attenuates NF-kappaB signaling and stabilizes latent virus persistence (Q24648180)

5

object named as

Carlo M Croce

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author name string

Fang Lu

1

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Andreas Weidmer

2

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Chang-Gong Liu

3

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Paul M Lieberman

MicroRNA-155 is an Epstein-Barr virus-induced gene that modulates Epstein-Barr virus-regulated gene expression pathways

6

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language of work or name

English

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publication date

November 2008

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published in

Journal of Virology

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volume

82

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issue

21

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page(s)

10436-43

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cites work

1 reference

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B-cell receptor activation induces BIC/miR-155 expression through a conserved AP-1 element

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MicroRNA-155 is induced during the macrophage inflammatory response

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bic, a novel gene activated by proviral insertions in avian leukosis virus-induced lymphomas, is likely to function through its noncoding RNA

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Integrating cell-signalling pathways with NF-kappaB and IKK function

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7 April 2017

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Triggering the interferon antiviral response through an IKK-related pathway

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Regulation of the germinal center response by microRNA-155

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microRNA-155 regulates the generation of immunoglobulin class-switched plasma cells

7 April 2017

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Modulation of miR-155 and miR-125b levels following lipopolysaccharide/TNF-alpha stimulation and their possible roles in regulating the response to endotoxin shock

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BIC and miR-155 are highly expressed in Hodgkin, primary mediastinal and diffuse large B cell lymphomas

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Epstein-Barr virus-encoded latent membrane protein 1 (LMP1) induces the expression of the cellular microRNA miR-146a

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MicroRNA-155 function in B Cells

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Latent membrane protein 1 of Epstein-Barr virus coordinately regulates proliferation with control of apoptosis.

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