Soluble TNF mediates the transition from pulmonary inflammation to fibrosis (Q27303153)

7

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author name string

Nikos Oikonomou

1

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Vaggelis Harokopos

2

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Jonathan Zalevsky

3

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Christos Valavanis

4

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Anastasia Kotanidou

5

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Vassilis Aidinis

Creative Commons Attribution 4.0 International

8

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language of work or name

English

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publication date

2006

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published in

PLOS One

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volume

1

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issue

1

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page(s)

e108

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copyright license

start time

27 December 2006

1 reference

April 2022 Public Data File from Crossref

copyright status

copyrighted

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T cell independence of bleomycin-induced pulmonary fibrosis

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Contribution of T lymphocytes to the development of bleomycin-induced pulmonary fibrosis

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Abrogation of bleomycin-induced epithelial apoptosis and lung fibrosis by captopril or by a caspase inhibitor

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Repression of bleomycin-induced pneumopathy by TNF

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Treatment by human recombinant soluble TNF receptor of pulmonary fibrosis induced by bleomycin or silica in mice

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Transmembrane TNF is sufficient to initiate cell migration and granuloma formation and provide acute, but not long-term, control of Mycobacterium tuberculosis infection

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Overexpression of tumor necrosis factor-alpha produces an increase in lung volumes and pulmonary hypertension

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TNF-alpha-mediated lung cytokine networking and eosinophil recruitment in pulmonary fibrosis

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10.1371/JOURNAL.PONE.0000108

Identifiers

DOI

1 reference

ADS bibcode

2006PLoSO...1..108O

0 references

1 reference

1 reference

PubMed publication ID

17205112

1 reference