Hepatitis C Virus (HCV) Proteins Induce NADPH Oxidase 4 Expression in a Transforming Growth Factor -Dependent Manner: a New Contributor to HCV-Induced Oxidative Stress (Q27490473)

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English	Hepatitis C Virus (HCV) Proteins Induce NADPH Oxidase 4 Expression in a Transforming Growth Factor -Dependent Manner: a New Contributor to HCV-Induced Oxidative Stress	scientific article

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scholarly article

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Hepatitis C Virus (HCV) Proteins Induce NADPH Oxidase 4 Expression in a Transforming Growth Factor -Dependent Manner: a New Contributor to HCV-Induced Oxidative Stress (English)

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Hepatitis C virus

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based on heuristic

inferred from title

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based on heuristic

inferred from title

Suzanne U. Emerson

2

object named as

Suzanne U Emerson

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author name string

Howard E Boudreau

1

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Agnieszka Korzeniowska

3

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Meghan A Jendrysik

4

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Thomas L Leto

5

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publication date

December 2009

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Journal of Virology

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83

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24

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12934-46

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Genes involved in viral carcinogenesis and tumor initiation in hepatitis C virus-induced hepatocellular carcinoma

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Microarray analysis identifies a common set of cellular genes modulated by different HCV replicon clones

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Hepatitis C virus proteins

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Hepatitis C virus core protein promotes proliferation of human hepatoma cells through enhancement of transforming growth factor expression via activation of nuclear factor- B

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NAD(P)H oxidase Nox-4 mediates 7-ketocholesterol-induced endoplasmic reticulum stress and apoptosis in human aortic smooth muscle cells

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The NAD(P)H oxidase homolog Nox4 modulates insulin-stimulated generation of H2O2 and plays an integral role in insulin signal transduction

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Highly permissive cell lines for subgenomic and genomic hepatitis C virus RNA replication

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Identification of renox, an NAD(P)H oxidase in kidney

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Replication of hepatitis C virus

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Functional analysis of Nox4 reveals unique characteristics compared to other NADPH oxidases

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TGF-beta-induced epithelial to mesenchymal transition

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Regulation of ROS signal transduction by NADPH oxidase 4 localization

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Redox regulation of hepatitis C in nonalcoholic and alcoholic liver

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NOX in liver fibrosis

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Transforming growth factor-beta1 induces an epithelial-to-mesenchymal transition state in mouse hepatocytes in vitro

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HCV core protein induces hepatic lipid accumulation by activating SREBP1 and PPARgamma

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https://pubmed.ncbi.nlm.nih.gov/19812163

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based on heuristic

inferred from PubMed ID database lookup

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1 reference

https://pubmed.ncbi.nlm.nih.gov/19812163

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based on heuristic

inferred from PubMed ID database lookup

Hepatocyte steatosis in HCV patients promotes fibrosis by enhancing TGF-beta liver expression

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https://pubmed.ncbi.nlm.nih.gov/19812163

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inferred from PubMed ID database lookup

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1 reference

https://pubmed.ncbi.nlm.nih.gov/19812163

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1128/JVI.01059-09

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Consolidated OpenCitations Corpus – April 2017

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OpenCitations bibliographic resource ID

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Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

1 reference

Consolidated OpenCitations Corpus – April 2017

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