MET amplification leads to gefitinib resistance in lung cancer by activating ERBB3 signaling (Q27851406)

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English	MET amplification leads to gefitinib resistance in lung cancer by activating ERBB3 signaling	scientific article

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13 November 2016

MET amplification leads to gefitinib resistance in lung cancer by activating ERBB3 signaling (English)

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13 November 2016

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1 reference

14

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Tetsuya Mitsudomi

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3

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Lewis C. Cantley

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18

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Pasi A Jänne

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19

object named as

Pasi A Jänne

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Tony Mok

object named as

Tony Mok

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15

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stated in

ORCID Public Data File 2021

author name string

Jeffrey A Engelman

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1

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Kreshnik Zejnullahu

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2

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Youngchul Song

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4

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Courtney Hyland

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5

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Joon Oh Park

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6

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Neal Lindeman

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7

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Christopher-Michael Gale

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8

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Xiaojun Zhao

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9

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James Christensen

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10

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Takayuki Kosaka

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11

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Alison J Holmes

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12

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Andrew M Rogers

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13

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Charles Lee

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16

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Bruce E Johnson

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17

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language of work or name

English

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publication date

18 May 2007

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published in

Science

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volume

316

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page(s)

1039-43

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issue

5827

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describes a project that uses

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Allelic dilution obscures detection of a biologically significant resistance mutation in EGFR-amplified lung cancer

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Analysis of epidermal growth factor receptor gene mutation in patients with non-small cell lung cancer and acquired resistance to gefitinib.

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ErbB-3 mediates phosphoinositide 3-kinase activity in gefitinib-sensitive non-small cell lung cancer cell lines

1 reference

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Amplification of MET may identify a subset of cancers with extreme sensitivity to the selective tyrosine kinase inhibitor PHA-665752

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Activity of a specific inhibitor of the BCR-ABL tyrosine kinase in the blast crisis of chronic myeloid leukemia and acute lymphoblastic leukemia with the Philadelphia chromosome

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Molecular correlates of imatinib resistance in gastrointestinal stromal tumors.

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Inhibition of the met receptor in mesothelioma

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Interaction between Gab1 and the c-Met receptor tyrosine kinase is responsible for epithelial morphogenesis

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Prospective phase II study of gefitinib for chemotherapy-naive patients with advanced non-small-cell lung cancer with epidermal growth factor receptor gene mutations

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BCR-ABL independence and LYN kinase overexpression in chronic myelogenous leukemia cells selected for resistance to STI571.

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Molecular and chromosomal mechanisms of resistance to imatinib (STI571) therapy

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Mutations of the epidermal growth factor receptor gene in lung cancer: biological and clinical implications.

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Novel D761Y and common secondary T790M mutations in epidermal growth factor receptor-mutant lung adenocarcinomas with acquired resistance to kinase inhibitors

MET amplification leads to gefitinib resistance in lung cancer by activating ERBB3 signaling (Q27851406)

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MET amplification leads to gefitinib resistance in lung cancer by activating ERBB3 signaling (Q27851406)

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