Docetaxel-induced polyploidization may underlie chemoresistance and disease relapse (Q28087331)

main subject

whole genome duplication

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Angela Ogden

1

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Padmashree C.G. Rida

2

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Beatrice S. Knudsen

3

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Omer Kucuk

4

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Ritu Aneja

5

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language of work or name

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28 October 2015

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367

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issue

2

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page(s)

89-92

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Stem cells, senescence, neosis and self-renewal in cancer

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Phase III study of doxorubicin/cyclophosphamide with concomitant versus sequential docetaxel as adjuvant treatment in patients with human epidermal growth factor receptor 2-normal, node-positive breast cancer: BCIRG-005 trial

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Drug-induced senescence generates chemoresistant stemlike cells with low reactive oxygen species

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Illicit survival of cancer cells during polyploidization and depolyploidization

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Selective resistance of tetraploid cancer cells against DNA damage-induced apoptosis

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Neosis--a paradigm of self-renewal in cancer

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Secondary leukaemia after cure for locally advanced NSCLC: alkylating type secondary leukaemia after induction therapy with docetaxel and carboplatin for NSCLC IIIB.

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27 November 2018

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P27KiP1 overexpression inhibits the growth and doxorubicin sensitivity of HT29 human colon cancer cells in vivo

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1 reference

https://pubmed.ncbi.nlm.nih.gov/26185000

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Identifiers

DOI

10.1016/J.CANLET.2015.06.025

0 references

1 reference

PubMed publication ID

26185000

1 reference