Transcriptional repression of the E2F-1 gene by interferon-alpha is mediated through induction of E2F-4/pRB and E2F-4/p130 complexes (Q28141867)

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S Iwase

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J Kikuchi

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M Nakamura

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H Yamada

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M Matsuda

Activation of DNA-binding activity in an apparently cytoplasmic precursor of the NF-kappa B transcription factor

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language of work or name

English

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publication date

18 March 1999

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published in

Oncogene

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volume

18

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issue

11

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page(s)

2003-14

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cites work

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Domains A and B in the Rb pocket interact to form a transcriptional repressor motif

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A role for a bent DNA structure in E2F-mediated transcription activation

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Cellular targets for activation by the E2F1 transcription factor include DNA synthesis- and G1/S-regulatory genes

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Close link between reduction of c-myc expression by interferon and, G0/G1 arrest

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Structure and cell cycle-regulated transcription of the human cyclin A gene

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E2F-5, a new E2F family member that interacts with p130 in vivo

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The retinoblastoma gene product protects E2F-1 from degradation by the ubiquitin-proteasome pathway

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Autoregulatory control of E2F1 expression in response to positive and negative regulators of cell cycle progression.

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E2F4-RB and E2F4-p107 complexes suppress gene expression by transforming growth factor beta through E2F binding sites

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Interferons and interleukin-6 suppress the DNA-binding activity of E2F in growth-sensitive hematopoietic cells

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E2F-4 switches from p130 to p107 and pRB in response to cell cycle reentry

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Transcription of the E2F-1 gene is rendered cell cycle dependent by E2F DNA-binding sites within its promoter

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E2F: a link between the Rb tumor suppressor protein and viral oncoproteins

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Expression of the HsOrc1 gene, a human ORC1 homolog, is regulated by cell proliferation via the E2F transcription factor

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Interferons and interleukin 6 suppress phosphorylation of the retinoblastoma protein in growth-sensitive hematopoietic cells

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E2F-4 and E2F-5, two members of the E2F family, are expressed in the early phases of the cell cycle

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The accumulation of an E2F-p130 transcriptional repressor distinguishes a G0 cell state from a G1 cell state.

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Subunit composition determines E2F DNA-binding site specificity

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In vivo structure of the human cdc2 promoter: release of a p130-E2F-4 complex from sequences immediately upstream of the transcription initiation site coincides with induction of cdc2 expression

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Mechanism of active transcriptional repression by the retinoblastoma protein

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The predominant E2F complex in human primary haemopoietic cells and in AML blasts contains E2F-4, DP-1 and p130

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Cell cycle regulation by the retinoblastoma family of growth inhibitory proteins

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Transcriptional activation of the cdc2 gene is associated with Fas-induced apoptosis of human hematopoietic cells

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Modulation of E2F activity is linked to interferon-induced growth suppression of hematopoietic cells

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Expression cloning of a cDNA encoding a retinoblastoma-binding protein with E2F-like properties

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A novel CCAAT-binding protein necessary for adhesion-dependent cyclin A transcription at the G1/S boundary is sequestered by a retinoblastoma-like protein in G0

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A simple phase-extraction assay for chloramphenicol acyltransferase activity

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Functional interaction between E2F-4 and p130: evidence for distinct mechanisms underlying growth suppression by different retinoblastoma protein family members

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Changes of cell cycle-regulating genes in interferon-treated Daudi cells

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