alphaPIX nucleotide exchange factor is activated by interaction with phosphatidylinositol 3-kinase (Q28145949)

1

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M Tanaka

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Y Otsuki

3

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D Y Wang

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R J Guo

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Y Zhu

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R Takeda

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H Hanai

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E Kaneko

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H Sugimura

Wortmannin is a potent phosphatidylinositol 3-kinase inhibitor: the role of phosphatidylinositol 3,4,5-trisphosphate in neutrophil responses.

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language of work or name

English

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publication date

7 October 1999

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published in

Oncogene

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volume

18

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issue

41

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page(s)

5680-90

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cites work

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Human Ste20 homologue hPAK1 links GTPases to the JNK MAP kinase pathway

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Phosphorylation of tyrosine 397 in focal adhesion kinase is required for binding phosphatidylinositol 3-kinase

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Membrane targeting of p21-activated kinase 1 (PAK1) induces neurite outgrowth from PC12 cells

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alphaPix stimulates p21-activated kinase activity through exchange factor-dependent and -independent mechanisms

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The lipid products of phosphoinositide 3-kinase increase cell motility through protein kinase C.

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The adaptor protein Nck links receptor tyrosine kinases with the serine-threonine kinase Pak1

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Signaling through CD5 activates a pathway involving phosphatidylinositol 3-kinase, Vav, and Rac1 in human mature T lymphocytes

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1-Phosphatidylinositol 3-kinase activity is required for insulin-stimulated glucose transport but not for RAS activation in CHO cells

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Membrane localization of phosphatidylinositol 3-kinase is sufficient to activate multiple signal-transducing kinase pathways

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Activation of Pak by membrane localization mediated by an SH3 domain from the adaptor protein Nck.

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PAK kinases are directly coupled to the PIX family of nucleotide exchange factors

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The SH2/SH3 domain-containing protein Nck is recognized by certain anti-phospholipase C-gamma 1 monoclonal antibodies, and its phosphorylation on tyrosine is stimulated by platelet-derived growth factor and epidermal growth factor treatment

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Two signaling molecules share a phosphotyrosine-containing binding site in the platelet-derived growth factor receptor

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Rho family GTPases regulate p38 mitogen-activated protein kinase through the downstream mediator Pak1.

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