Pim kinases are upregulated during Epstein-Barr virus infection and enhance EBNA2 activity (Q28235877)

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author name string

Eeva-Marja Rainio

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Kara L Carter

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Marja Ruuska

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Sampsa Matikainen

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Päivi J Koskinen

Early events in Epstein-Barr virus infection of human B lymphocytes

7

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language of work or name

English

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publication date

15 March 2005

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published in

Virology

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volume

333

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issue

2

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page(s)

201-6

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cites work

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2005.01.001

Pim-1 kinase promotes inactivation of the pro-apoptotic Bad protein by phosphorylating it on the Ser112 gatekeeper site

7 January 2021

1 reference

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Pim-2 transgene induces lymphoid tumors, exhibiting potent synergy with c-myc

7 January 2021

1 reference

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The human protooncogene product p33pim is expressed during fetal hematopoiesis and in diverse leukemias

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2005.01.001

Epstein-barr virus transformation: involvement of latent membrane protein 1-mediated activation of NF-kappaB.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2005.01.001

The human EBNA-2 coactivator p100: multidomain organization and relationship to the staphylococcal nuclease fold and to the tudor protein involved in Drosophila melanogaster development

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2005.01.001

Epstein-barr virus-induced changes in B-lymphocyte gene expression

7 January 2021

1 reference

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A micrococcal nuclease homologue in RNAi effector complexes

7 January 2021

1 reference

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Pim serine/threonine kinases regulate the stability of Socs-1 protein

7 January 2021

1 reference

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The serine/threonine kinase Pim-2 is a transcriptionally regulated apoptotic inhibitor

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2005.01.001

Protein kinase A associates with HA95 and affects transcriptional coactivation by Epstein-Barr virus nuclear proteins

7 January 2021

1 reference

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Pim-1 kinase and p100 cooperate to enhance c-Myb activity

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2005.01.001

The PIM-1 serine kinase prolongs survival and inhibits apoptosis-related mitochondrial dysfunction in part through a bcl-2-dependent pathway

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2005.01.001

Cytokine signaling and Epstein-Barr virus-mediated cell transformation

7 January 2021

1 reference

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Pim-1 kinase inhibits STAT5-dependent transcription via its interactions with SOCS1 and SOCS3.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2005.01.001

Cutting edge: Transcriptional activity of NFATc1 is enhanced by the Pim-1 kinase

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2005.01.001

The Epstein-Barr virus nuclear protein 2 acidic domain forms a complex with a novel cellular coactivator that can interact with TFIIE

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2005.01.001

Predisposition to lymphomagenesis in pim-1 transgenic mice: cooperation with c-myc and N-myc in murine leukemia virus-induced tumors

7 January 2021

1 reference

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The PIM-2 kinase phosphorylates BAD on serine 112 and reverses BAD-induced cell death

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2005.01.001

Expression of Epstein-Barr virus transformation-associated genes in tissues of patients with EBV lymphoproliferative disease

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2005.01.001

10.1016/J.VIROL.2005.01.001

7 January 2021

Identifiers

DOI

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Consolidated OpenCitations Corpus – April 2017

release_tykklygq5bgc5a53hjj6oqzep4

4850887

Fatcat ID

1 reference

Fatcat

https://api.fatcat.wiki/v0/release/tykklygq5bgc5a53hjj6oqzep4

mapped directly with Wikidata item

24 November 2022

based on heuristic

4850887

1 reference

Consolidated OpenCitations Corpus – April 2017

Consolidated OpenCitations Corpus – April 2017

1 reference