Pim-1 kinase and p100 cooperate to enhance c-Myb activity (Q28288284)

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P J Koskinen

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F C Orrico

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E M Rainio

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K J Jalkanen

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A B Dash

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R N Eisenman

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S A Ness

The human protooncogene product p33pim is expressed during fetal hematopoiesis and in diverse leukemias

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language of work or name

English

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publication date

October 1998

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published in

Molecular Cell

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volume

2

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issue

4

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page(s)

417-25

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cites work

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The EVES motif mediates both intermolecular and intramolecular regulation of c-Myb

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Carboxy-terminal elements of c-Myb negatively regulate transcriptional activation in cis and in trans

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The retinoblastoma protein associates with the protein phosphatase type 1 catalytic subunit

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Leukemogenesis: Small differences in Myb have large effects

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c-Maf interacts with c-Myb to regulate transcription of an early myeloid gene during differentiation

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Point mutations in v-Myb disrupt a cyclophilin-catalyzed negative regulatory mechanism

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Ubiquitous expression and cell cycle regulation of the protein kinase PIM-1.

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Myb DNA binding inhibited by phosphorylation at a site deleted during oncogenic activation

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The human Pim-1 gene is selectively transcribed in different hemato-lymphoid cell lines in spite of a G + C-rich housekeeping promoter

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Molecular cloning reveals that the p160 Myb-binding protein is a novel, predominantly nucleolar protein which may play a role in transactivation by Myb

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