A second Ewing's sarcoma translocation, t(21;22), fuses the EWS gene to another ETS-family transcription factor, ERG (Q28252253)

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S L Lessnick

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D Lopez-Terrada

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X F Liu

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T J Triche

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C T Denny

https://scigraph.springernature.com/pub.10.1038/ng0294-146

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language of work or name

English

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publication date

February 1994

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published in

Nature Genetics

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volume

6

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issue

2

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page(s)

146-51

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exact match

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cites work

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Single-step purification of polypeptides expressed in Escherichia coli as fusions with glutathione S-transferase

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Chromosome translocation in peripheral neuroepithelioma

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Ewing sarcoma 11;22 translocation produces a chimeric transcription factor that requires the DNA-binding domain encoded by FLI1 for transformation.

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Erythroleukemia induction by Friend murine leukemia virus: insertional activation of a new member of the ets gene family, Fli-1, closely linked to c-ets-1.

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The Ets family of transcription factors

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The Ewing's sarcoma EWS/FLI-1 fusion gene encodes a more potent transcriptional activator and is a more powerful transforming gene than FLI-1

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erg, a human ets-related gene on chromosome 21: alternative splicing, polyadenylation, and translation

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Evolutionarily conserved Ets family members display distinct DNA binding specificities

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Chromosomal translocation t(1;19) results in synthesis of a homeobox fusion mRNA that codes for a potential chimeric transcription factor

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A new homeobox gene contributes the DNA binding domain of the t(1;19) translocation protein in pre-B ALL

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Fusion of the leucine zipper gene HLF to the E2A gene in human acute B-lineage leukemia

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Genes on chromosomes 4, 9, and 19 involved in 11q23 abnormalities in acute leukemia share sequence homology and/or common motifs

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Acute mixed-lineage leukemia t(4;11)(q21;q23) generates an MLL-AF4 fusion product

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High-resolution mapping of human chromosome 11 by in situ hybridization with cosmid clones

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