Mouse hesr1 and hesr2 genes are redundantly required to mediate Notch signaling in the developing cardiovascular system (Q28304868)

Hairy/enhancer-of-split related with YRPW motif 2

GOA release 2020-03-11

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GOA release 2020-03-11

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5

Randy L Johnson

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author name string

Hiroki Kokubo

1

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Sachiko Miyagawa-Tomita

2

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Makoto Nakazawa

3

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Yumiko Saga

Roles of ephrinB ligands and EphB receptors in cardiovascular development: demarcation of arterial/venous domains, vascular morphogenesis, and sprouting angiogenesis

4

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language of work or name

English

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publication date

15 February 2005

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published in

Developmental Biology

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volume

278

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issue

2

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page(s)

301-9

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cites work

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Regulation of chamber-specific gene expression in the developing heart by Irx4.

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Hey genes in cardiovascular development

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Symmetrical mutant phenotypes of the receptor EphB4 and its specific transmembrane ligand ephrin-B2 in cardiovascular development

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NK-2 homeobox genes and heart development

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Embryonic lethality in mice homozygous for a processing-deficient allele of Notch1.

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HERP1 is a cell type-specific primary target of Notch

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Notch signaling in vascular development

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Synergy and antagonism between Notch and BMP receptor signaling pathways in endothelial cells.

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Expression and function of bone morphogenetic proteins in the development of the embryonic endocardial cushions

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Targeted disruption of hesr2 results in atrioventricular valve anomalies that lead to heart dysfunction

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Alagille syndrome is caused by mutations in human Jagged1, which encodes a ligand for Notch1

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Myogenic and morphogenetic defects in the heart tubes of murine embryos lacking the homeo box gene Nkx2-5

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Pathological study of Japanese quail embryo with acid alpha-glucosidase deficiency during early development

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Mutations in the human Jagged1 gene are responsible for Alagille syndrome

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Mechanisms of angiogenesis

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Invasion of mesenchyme into three-dimensional collagen gels: a regional and temporal analysis of interaction in embryonic heart tissue

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A subclass of bHLH proteins required for cardiac morphogenesis

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Regulation of cardiac mesodermal and neural crest development by the bHLH transcription factor, dHAND

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Bone morphogenetic protein-2 can mediate myocardial regulation of atrioventricular cushion mesenchymal cell formation in mice

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Enhanced gene activation by Notch and BMP signaling cross-talk

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Notch promotes epithelial-mesenchymal transition during cardiac development and oncogenic transformation

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Vascular expression of Notch pathway receptors and ligands is restricted to arterial vessels.

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Molecular distinction and angiogenic interaction between embryonic arteries and veins revealed by ephrin-B2 and its receptor Eph-B4

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Embryonic lethality and vascular defects in mice lacking the Notch ligand Jagged1

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The basic helix-loop-helix transcription factor, dHAND, is required for vascular development

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gridlock, an HLH gene required for assembly of the aorta in zebrafish

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Gridlock signalling pathway fashions the first embryonic artery

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