Norcantharidin-induced apoptosis is via the extracellular signal-regulated kinase and c-Jun-NH2-terminal kinase signaling pathways in human hepatoma HepG2 cells (Q28391230)

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English	Norcantharidin-induced apoptosis is via the extracellular signal-regulated kinase and c-Jun-NH2-terminal kinase signaling pathways in human hepatoma HepG2 cells	scientific article

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Europe PubMed Central

Norcantharidin-induced apoptosis is via the extracellular signal-regulated kinase and c-Jun-NH2-terminal kinase signaling pathways in human hepatoma HepG2 cells (English)

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hepatocellular carcinoma

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inferred from title

author name string

Yan-Nian Chen

1

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Chi-Chih Cheng

2

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Jung-Chou Chen

3

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Wei Tsauer

4

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Shih-Lan Hsu

5

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language of work or name

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publication date

October 2003

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British Journal of Pharmacology

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140

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3

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461-70

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New insights into the role of nuclear factor-kB in cell growth regulation

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From receptors to stress-activated MAP kinases

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20 March 2017

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Embryonic lethality and liver degeneration in mice lacking the RelA component of NF-κB

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Opposing effects of ERK and JNK-p38 MAP kinases on apoptosis

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AP-1 in cell proliferation and survival

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The JNK signal transduction pathway

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Generation of reactive oxygen intermediates, activation of NF-kappaB, and induction of apoptosis in human endothelial cells by glucose: role of nitric oxide synthase?

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Arsenic induces apoptosis through a c-Jun NH2-terminal kinase-dependent, p53-independent pathway.

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NF-kappaB to the rescue: RELs, apoptosis and cellular transformation

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Requirement for ceramide-initiated SAPK/JNK signalling in stress-induced apoptosis.

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Nonradioactive gel mobility shift assay using chemiluminescent detection

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The role of c-Jun N-terminal kinase (JNK) in apoptosis induced by ultraviolet C and gamma radiation. Duration of JNK activation may determine cell death and proliferation

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The extracellular signal-regulated kinase pathway is required for activation-induced cell death of T cells

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Identifiers

10.1038/SJ.BJP.0705461

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