MyD88-, but not Nod1- and/or Nod2-deficient mice, show increased susceptibility to polymicrobial sepsis due to impaired local inflammatory response (Q28512466)

From Wikidata

Jump to navigation Jump to search

scientific journal article

Language	Label	Description	Also known as
English	MyD88-, but not Nod1- and/or Nod2-deficient mice, show increased susceptibility to polymicrobial sepsis due to impaired local inflammatory response	scientific journal article

Statements

scholarly article

1 reference

25 January 2017

MyD88-, but not Nod1- and/or Nod2-deficient mice, show increased susceptibility to polymicrobial sepsis due to impaired local inflammatory response (English)

1 reference

25 January 2017

0 references

0 references

Chemokine (C-X-C motif) ligand 2

1 reference

GOA release 2020-03-11

1 reference

GOA release 2020-03-11

Tumor necrosis factor

1 reference

GOA release 2020-03-11

Chemokine (C-X-C motif) ligand 1

1 reference

GOA release 2020-03-11

Myeloid differentiation primary response gene 88

1 reference

GOA release 2020-03-11

Fernando Queiroz Cunha

object named as

Fernando Q. Cunha

13

0 references

Fernando Spiller

object named as

Fernando Spiller

9

0 references

Andressa Freitas

object named as

Andressa Freitas

8

0 references

Alexandre Kanashiro

object named as

Alexandre Kanashiro

4

0 references

Dario S. Zamboni

object named as

Dario S. Zamboni

11

0 references

author name string

Fabiane Sônego

1

1 reference

25 January 2017

Fernanda V. S. Castanheira

2

1 reference

25 January 2017

Paula G. Czaikoski

3

1 reference

25 January 2017

Fabricio O. Souto

5

1 reference

25 January 2017

Rafael O. França

6

1 reference

25 January 2017

Daniele C. Nascimento

7

1 reference

25 January 2017

Larissa D. Cunha

10

1 reference

25 January 2017

José C. Alves-Filho

12

1 reference

25 January 2017

language of work or name

0 references

publication date

1 January 2014

1 reference

25 January 2017

1 reference

25 January 2017

9

1 reference

25 January 2017

8

1 reference

25 January 2017

e103734

1 reference

25 January 2017

copyright license

Creative Commons Attribution 4.0 International

1 August 2014

1 reference

April 2022 Public Data File from Crossref

copyright status

0 references

Inflection points in sepsis biology: from local defense to systemic organ injury

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4118952

19 March 2017

TLR2, TLR4 and the MYD88 signaling pathway are crucial for neutrophil migration in acute kidney injury induced by sepsis

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4118952

19 March 2017

Neutrophil secretion products regulate anti-bacterial activity in monocytes and macrophages

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4118952

19 March 2017

Signalling pathways and molecular interactions of NOD1 and NOD2

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4118952

19 March 2017

NLRs join TLRs as innate sensors of pathogens

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4118952

19 March 2017

Nod2, a Nod1/Apaf-1 family member that is restricted to monocytes and activates NF-kappaB

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4118952

19 March 2017

Toll-like receptors and their crosstalk with other innate receptors in infection and immunity

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4118952

7 April 2017

The immunopathogenesis of sepsis

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4118952

7 April 2017

NOD2-mediated suppression of CD55 on neutrophils enhances C5a generation during polymicrobial sepsis

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4118952

29 September 2017

NOD1 and NOD2 Signaling in Infection and Inflammation.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4118952

29 September 2017

MyD88 and Trif signaling play distinct roles in cardiac dysfunction and mortality during endotoxin shock and polymicrobial sepsis.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4118952

29 September 2017

Cecal ligation and puncture: the gold standard model for polymicrobial sepsis?

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4118952

29 September 2017

Neutrophil paralysis in sepsis

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4118952

29 September 2017

IL-17 receptor signaling is required to control polymicrobial sepsis

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4118952

29 September 2017

The NOD/RIP2 pathway is essential for host defenses against Chlamydophila pneumoniae lung infection

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4118952

29 September 2017

Regulation of chemokine receptor by Toll-like receptor 2 is critical to neutrophil migration and resistance to polymicrobial sepsis

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4118952

29 September 2017

Muramyldipeptide modulates CXCL-8 release of BEAS-2B cells via NOD2.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4118952

29 September 2017

Critical role of toll-like receptors and nucleotide oligomerisation domain in the regulation of health and disease

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4118952

29 September 2017

Nod1 acts as an intracellular receptor to stimulate chemokine production and neutrophil recruitment in vivo

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4118952

29 September 2017

Induction of Nod1 and Nod2 intracellular pattern recognition receptors in murine osteoblasts following bacterial challenge.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4118952

29 September 2017

Sepsis and septic shock--a review of laboratory models and a proposal

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4118952

29 September 2017

Neutrophil migration during liver injury is under nucleotide-binding oligomerization domain 1 control

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4118952

2 June 2018

Toll-like receptor 9 activation in neutrophils impairs chemotaxis and reduces sepsis outcome.

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0103734

21 January 2018

Deletion of MyD88 markedly attenuates sepsis-induced T and B lymphocyte apoptosis but worsens survival.

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0103734

21 January 2018

Mutations of genes involved in the innate immune system as predictors of sepsis in very low birth weight infants

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0103734

21 January 2018

Failure of neutrophil chemotactic function in septic patients.

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0103734

21 January 2018

The expression and function of Nod-like receptors in neutrophils

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0103734

21 January 2018

The pattern recognition receptors Nod1 and Nod2 account for neutrophil recruitment to the lungs of mice infected with Legionella pneumophila

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0103734

21 January 2018

Nod1/RICK and TLR signaling regulate chemokine and antimicrobial innate immune responses in mesothelial cells.

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0103734

21 January 2018

Toll-like receptor 4 signaling leads to neutrophil migration impairment in polymicrobial sepsis.

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0103734

21 January 2018

Cutting edge: Divergent cell-specific functions of MyD88 for inflammatory responses and organ injury in septic peritonitis.

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0103734

21 January 2018

Selective NOD1 Agonists Cause Shock and Organ Injury/DysfunctionIn Vivo

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0103734

21 January 2018

Muramyldipeptide and diaminopimelic acid-containing desmuramylpeptides in combination with chemically synthesized Toll-like receptor agonists synergistically induced production of interleukin-8 in a NOD2- and NOD1-dependent manner, respectively, in h

1 reference

https://pubmed.ncbi.nlm.nih.gov/25084278

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

New biomarkers sought for improving sepsis management and care

1 reference

https://pubmed.ncbi.nlm.nih.gov/25084278

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Role of nitric oxide in the failure of neutrophil migration in sepsis

1 reference

https://pubmed.ncbi.nlm.nih.gov/25084278

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Cutting edge: myeloid differentiation factor 88 deficiency improves resistance against sepsis caused by polymicrobial infection

1 reference

https://pubmed.ncbi.nlm.nih.gov/25084278

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Genetic background determines susceptibility during murine septic peritonitis

1 reference

https://pubmed.ncbi.nlm.nih.gov/25084278

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Improved host defense against septic peritonitis in mice lacking MyD88 and TRIF is linked to a normal interferon response

1 reference

https://pubmed.ncbi.nlm.nih.gov/25084278

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1371/JOURNAL.PONE.0103734

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

2014PLoSO...9j3734S

0 references

OpenCitations bibliographic resource ID

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

ResearchGate publication ID

0 references

Sitelinks

Wikipedia(0 entries)

Wikibooks(0 entries)

Wikinews(0 entries)

Wikiquote(0 entries)

Wikisource(0 entries)

Wikiversity(0 entries)

Wikivoyage(0 entries)

Wiktionary(0 entries)

Multilingual sites(0 entries)

Retrieved from "https://www.wikidata.org/w/index.php?title=Q28512466&oldid=1860990171"