Cytochrome c deficiency causes embryonic lethality and attenuates stress-induced apoptosis (Q28512752)

GOA release 2020-03-11

cytochrome c, somatic

1 reference

GOA release 2020-03-11

author name string

K. Li

1

1 reference

25 January 2017

Y. Li

2

1 reference

25 January 2017

J. M. Shelton

3

1 reference

25 January 2017

J. A. Richardson

4

1 reference

25 January 2017

E. Spencer

5

1 reference

25 January 2017

Z. J. Chen

6

1 reference

25 January 2017

X. Wang

7

1 reference

25 January 2017

R. S. Williams

describes a project that uses

8

1 reference

25 January 2017

language of work or name

English

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publication date

12 May 2000

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25 January 2017

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25 January 2017

101

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25 January 2017

389–399

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25 January 2017

4

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25 January 2017

Cyt c-/-

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Apaf1 (CED-4 homolog) regulates programmed cell death in mammalian development

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Differential requirement for caspase 9 in apoptotic pathways in vivo.

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Mitochondrial depolarization accompanies cytochrome c release during apoptosis in PC6 cells

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Bcl-2 blocks apoptosis in cells lacking mitochondrial DNA.

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Cytochrome c-mediated apoptosis in cells lacking mitochondrial DNA. Signaling pathway involving release and caspase 3 activation is conserved

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Cytochrome c activation of CPP32-like proteolysis plays a critical role in a Xenopus cell-free apoptosis system.

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Decreased apoptosis in the brain and premature lethality in CPP32-deficient mice

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Reduced apoptosis and cytochrome c-mediated caspase activation in mice lacking caspase 9

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Mitochondrial transcription factor A is necessary for mtDNA maintenance and embryogenesis in mice

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Cloning and characterization of three new murine genes encoding short homologues of RNase P RNA.

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Cell-specific induction of apoptosis by microinjection of cytochrome c. Bcl-xL has activity independent of cytochrome c release

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Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

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Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

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Stress-signalling kinase Sek1 protects thymocytes from apoptosis mediated by CD95 and CD3

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Dark is a Drosophila homologue of Apaf-1/CED-4 and functions in an evolutionarily conserved death pathway

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Apaf1 is required for mitochondrial pathways of apoptosis and brain development

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Injected cytochrome c induces apoptosis

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An APAF-1.cytochrome c multimeric complex is a functional apoptosome that activates procaspase-9

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Identifiers

DOI

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Consolidated OpenCitations Corpus – April 2017

release_7vapfw3h7ffpnjtkgim2nx3ele

1995099

Fatcat ID

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Fatcat

https://api.fatcat.wiki/v0/release/7vapfw3h7ffpnjtkgim2nx3ele

24 November 2022

mapped directly with Wikidata item

1995099

1 reference

Consolidated OpenCitations Corpus – April 2017

Consolidated OpenCitations Corpus – April 2017

1 reference