Growth factor and Th2 cytokine signaling pathways converge at STAT6 to promote arginase expression in progressive experimental visceral leishmaniasis (Q28540097)

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scientific article (publication date: June 2014)

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English	Growth factor and Th2 cytokine signaling pathways converge at STAT6 to promote arginase expression in progressive experimental visceral leishmaniasis	scientific article (publication date: June 2014)

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scholarly article

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Growth factor and Th2 cytokine signaling pathways converge at STAT6 to promote arginase expression in progressive experimental visceral leishmaniasis (English)

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visceral leishmaniasis

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based on heuristic

inferred from title

author name string

E Yaneth Osorio

1

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Bruno L Travi

2

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Alda M da Cruz

3

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Omar A Saldarriaga

4

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Audrie A Medina

5

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Peter C Melby

6

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language of work or name

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publication date

June 2014

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10

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6

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e1004165

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copyright license

Creative Commons Attribution 4.0 International

26 June 2014

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April 2022 Public Data File from Crossref

copyright status

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Leishmaniasis worldwide and global estimates of its incidence

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Interferon-gamma release assay (modified QuantiFERON) as a potential marker of infection for Leishmania donovani, a proof of concept study

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Identification of small molecule lead compounds for visceral leishmaniasis using a novel ex vivo splenic explant model system

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Local suppression of T cell responses by arginase-induced L-arginine depletion in nonhealing leishmaniasis

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Arginase I expression and activity in human mononuclear cells after injury

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IL-4/IL-13 signaling beyond JAK/STAT

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In vivo cytokine profiles in patients with kala-azar. Marked elevation of both interleukin-10 and interferon-gamma

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Alternative activation of macrophages: mechanism and functions

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Alternative activation of macrophages: an immunologic functional perspective

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The interactions and essential effects of intrinsic insulin-like growth factor-I on Leishmania (Leishmania) major growth within macrophages.

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Reassessment of immune correlates in human visceral leishmaniasis as defined by cytokine release in whole blood

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Progressive visceral leishmaniasis is driven by dominant parasite-induced STAT6 activation and STAT6-dependent host arginase 1 expression

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Myeloid cell IL-10 production in response to leishmania involves inactivation of glycogen synthase kinase-3β downstream of phosphatidylinositol-3 kinase.

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Compartment-specific remodeling of splenic micro-architecture during experimental visceral leishmaniasis.

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mTOR signaling pathway regulates the IL-12/IL-10 axis in Leishmania donovani infection

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29 September 2017

Arginase in parasitic infections: macrophage activation, immunosuppression, and intracellular signals

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Toll-like receptor-induced arginase 1 in macrophages thwarts effective immunity against intracellular pathogens

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29 September 2017

Splenic accumulation of IL-10 mRNA in T cells distinct from CD4+CD25+ (Foxp3) regulatory T cells in human visceral leishmaniasis

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29 September 2017

Leishmania promastigotes activate PI3K/Akt signalling to confer host cell resistance to apoptosis

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29 September 2017

Subversion mechanisms by which Leishmania parasites can escape the host immune response: a signaling point of view.

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Alternatively activated macrophages during parasite infections

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12 December 2020

based on heuristic

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https://pubmed.ncbi.nlm.nih.gov/24967908

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10.1371/JOURNAL.PPAT.1004165

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Consolidated OpenCitations Corpus – April 2017

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Consolidated OpenCitations Corpus – April 2017

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Consolidated OpenCitations Corpus – April 2017

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