Copper-induced regression of cardiomyocyte hypertrophy is associated with enhanced vascular endothelial growth factor receptor-1 signalling pathway (Q28575727)

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English	Copper-induced regression of cardiomyocyte hypertrophy is associated with enhanced vascular endothelial growth factor receptor-1 signalling pathway	scientific journal article

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scholarly article

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31 January 2017

Copper-induced regression of cardiomyocyte hypertrophy is associated with enhanced vascular endothelial growth factor receptor-1 signalling pathway (English)

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31 January 2017

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author name string

Yang Zhou

1

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31 January 2017

Katherine Bourcy

2

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31 January 2017

Y. James Kang

3

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31 January 2017

publication date

1 October 2009

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31 January 2017

Cardiovascular Research

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31 January 2017

84

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31 January 2017

1

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31 January 2017

54–63

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31 January 2017

Role of PlGF in the intra- and intermolecular cross talk between the VEGF receptors Flt1 and Flk1

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2741345

17 March 2017

Dietary reference intakes: vitamin A, vitamin K, arsenic, boron, chromium, copper, iodine, iron, manganese, molybdenum, nickel, silicon, vanadium, and zinc

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2741345

17 March 2017

The vascular endothelial growth factor receptor KDR activates multiple signal transduction pathways in porcine aortic endothelial cells

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2741345

17 March 2017

Different signal transduction properties of KDR and Flt1, two receptors for vascular endothelial growth factor

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2741345

17 March 2017

A unique signal transduction from FLT tyrosine kinase, a receptor for vascular endothelial growth factor VEGF

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2741345

17 March 2017

Dietary copper supplementation reverses hypertrophic cardiomyopathy induced by chronic pressure overload in mice

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2741345

29 September 2017

Vascular endothelial growth factor blockade promotes the transition from compensatory cardiac hypertrophy to failure in response to pressure overload

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2741345

29 September 2017

Inhibition of calcineurin-NFAT hypertrophy signaling by cGMP-dependent protein kinase type I in cardiac myocytes

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2741345

29 September 2017

Copper reverses cardiomyocyte hypertrophy through vascular endothelial growth factor-mediated reduction in the cell size

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2741345

3 June 2018

Cardiac growth and angiogenesis coordinated by intertissue interactions

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2741345

3 June 2018

Structure of a VEGF-VEGF receptor complex determined by electron microscopy

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2741345

3 June 2018

Disruption of coordinated cardiac hypertrophy and angiogenesis contributes to the transition to heart failure

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVP178

21 January 2018

Vascular permeability factor (VPF)/vascular endothelial growth factor (VEGF) peceptor-1 down-modulates VPF/VEGF receptor-2-mediated endothelial cell proliferation, but not migration, through phosphatidylinositol 3-kinase-dependent pathways

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVP178

21 January 2018

Carvedilol prevents cardiac hypertrophy and overexpression of hypoxia-inducible factor-1alpha and vascular endothelial growth factor in pressure-overloaded rat heart

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVP178

21 January 2018

Gene transfer of cGMP-dependent protein kinase I enhances the antihypertrophic effects of nitric oxide in cardiomyocytes.

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVP178

21 January 2018

Chronic inhibition of cyclic GMP phosphodiesterase 5A prevents and reverses cardiac hypertrophy

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVP178

21 January 2018

p53-induced inhibition of Hif-1 causes cardiac dysfunction during pressure overload

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVP178

21 January 2018

Elevated Flk1 (vascular endothelial growth factor receptor 2) signaling mediates enhanced angiogenesis in beta3-integrin-deficient mice

1 reference

https://pubmed.ncbi.nlm.nih.gov/19542178

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Vascular endothelial growth factor induces activation and subcellular translocation of focal adhesion kinase (p125FAK) in cultured rat cardiac myocytes

1 reference

https://pubmed.ncbi.nlm.nih.gov/19542178

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Contributory role of VEGF overexpression in endothelin-1-induced cardiomyocyte hypertrophy

1 reference

https://pubmed.ncbi.nlm.nih.gov/19542178

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Receptor chimeras indicate that the vascular endothelial growth factor receptor-1 (VEGFR-1) modulates mitogenic activity of VEGFR-2 in endothelial cells

1 reference

https://pubmed.ncbi.nlm.nih.gov/19542178

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

A Vascular Endothelial Growth Factor Antagonist Is Produced by the Human Placenta and Released into the Maternal Circulation1

1 reference

https://pubmed.ncbi.nlm.nih.gov/19542178

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1093/CVR/CVP178

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31 January 2017

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31 January 2017

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