Mechanisms contributing to the deficits in hippocampal synaptic plasticity in mice lacking amyloid precursor protein (Q28592893)

31 January 2017

title

Mechanisms contributing to the deficits in hippocampal synaptic plasticity in mice lacking amyloid precursor protein (English)

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Amyloid beta (A4) precursor protein

31 January 2017

main subject

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GOA release 2020-03-11

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G. R. Seabrook

1

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31 January 2017

D. W. Smith

2

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31 January 2017

B. J. Bowery

3

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31 January 2017

A. Easter

4

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31 January 2017

T. Reynolds

5

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31 January 2017

S. M. Fitzjohn

6

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31 January 2017

R. A. Morton

7

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31 January 2017

H. Zheng

8

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31 January 2017

G. R. Dawson

9

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31 January 2017

D. J. Sirinathsinghji

10

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31 January 2017

C. H. Davies

11

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31 January 2017

G. L. Collingridge

12

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31 January 2017

R. G. Hill

13

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31 January 2017

publication date

1 March 1999

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31 January 2017

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31 January 2017

volume

38

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31 January 2017

issue

3

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31 January 2017

page(s)

349–359

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Mutant presenilins of Alzheimer's disease increase production of 42-residue amyloid beta-protein in both transfected cells and transgenic mice

31 January 2017

cites work

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Amyloid precursor protein is localized in growing neurites of neonatal rat brain

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The amyloid protein precursor of Alzheimer's disease is a mediator of the effects of nerve growth factor on neurite outgrowth.

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The beta-amyloid precursor protein of Alzheimer's disease enhances neuron viability and modulates neuronal polarity

7 January 2021

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Cell-surface beta-amyloid precursor protein stimulates neurite outgrowth of hippocampal neurons in an isoform-dependent manner

7 January 2021

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7 January 2021

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beta-Amyloid precursor protein-deficient mice show reactive gliosis and decreased locomotor activity

7 January 2021

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10.1016/S0028-3908(98)00204-4

7 January 2021

Identifiers

DOI

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