Inactivation of Six2 in mouse identifies a novel genetic mechanism controlling development and growth of the cranial base (Q28593802)

Sine oculis-related homeobox 1

GOA release 2020-03-11

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Sine oculis-related homeobox 4

GOA release 2020-03-11

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GOA release 2020-03-11

author

Karen Piper Hanley

object named as

Karen Piper Hanley

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31 January 2017

Neil Hanley

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31 January 2017

8

Ward C

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31 January 2017

6

Raphaëlle Grifone

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31 January 2017

Guiyuan He

1

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31 January 2017

Sara Tavella

2

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31 January 2017

Michelle Self

4

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31 January 2017

Guillermo Oliver

5

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31 January 2017

Nicoletta Bobola

Interactions between Sox9 and beta-catenin control chondrocyte differentiation

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31 January 2017

language of work or name

English

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publication date

15 August 2010

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31 January 2017

Developmental Biology

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31 January 2017

344

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31 January 2017

2

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31 January 2017

720–730

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31 January 2017

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https://api.crossref.org/works/10.1016%2FJ.YDBIO.2010.05.509

Slc12a2 is a direct target of two closely related homeobox proteins, Six1 and Six4

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Gly369Cys mutation in mouse FGFR3 causes achondroplasia by affecting both chondrogenesis and osteogenesis

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A Ser(365)-->Cys mutation of fibroblast growth factor receptor 3 in mouse downregulates Ihh/PTHrP signals and causes severe achondroplasia

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The six family of homeobox genes in development and cancer

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Regulatory mechanisms in the pathways of cartilage and bone formation

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The family of bone morphogenetic proteins

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Misexpression of Six2 is associated with heritable frontonasal dysplasia and renal hypoplasia in 3H1 Br mice

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Hoxa-2 mutant mice exhibit homeotic transformation of skeletal elements derived from cranial neural crest

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Six proteins regulate the activation of Myf5 expression in embryonic mouse limbs

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Six1 and Six4 homeoproteins are required for Pax3 and Mrf expression during myogenesis in the mouse embryo

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Ectopic SOX9 mediates extracellular matrix deposition characteristic of organ fibrosis

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Dicer-dependent pathways regulate chondrocyte proliferation and differentiation

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Comparative three-dimensional analysis of CT-scans of the calvaria and cranial base in Apert and Crouzon syndromes

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Hoxa2 downregulates Six2 in the neural crest-derived mesenchyme

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Six2 functions redundantly immediately downstream of Hoxa2

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Altered myogenesis in Six1-deficient mice

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PTH/PTHrP receptor in early development and Indian hedgehog-regulated bone growth

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Anterior cranial base morphology in mice with midfacial retrusion.

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Segmental identity can change independently in the hindbrain and rhombencephalic neural crest.

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Mouse Otx2 functions in the formation and patterning of rostral head.

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Cranial base in craniofacial development: developmental features, influence on facial growth, anomaly, and molecular basis

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Plasticity of the endocranial base in nonsyndromic craniosynostosis

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Beta cell differentiation during early human pancreas development.

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Clinical and molecular cytogenetic characterisation of a newly recognised microdeletion syndrome involving 2p15-16.1

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Fgfr mRNA isoforms in craniofacial bone development.

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A homeotic transformation is generated in the rostral branchial region of the head by disruption of Hoxa-2, which acts as a selector gene

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Identification of transcriptional targets for Six5: implication for the pathogenesis of myotonic dystrophy type 1.

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Six2 activity is required for the formation of the mammalian pyloric sphincter

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Six2 is required for suppression of nephrogenesis and progenitor renewal in the developing kidney

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Cell proliferation inhibited by MyoD1 independently of myogenic differentiation

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C/EBPalpha is required to maintain postmitotic growth arrest in adipocytes

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CCAAT-enhancer binding protein: a component of a differentiation switch

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Regulation of rate of cartilage differentiation by Indian hedgehog and PTH-related protein

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Genetic control of skeletal development

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Indian and sonic hedgehogs regulate synchondrosis growth plate and cranial base development and function

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Three high mobility group-like sequences within a 48-base pair enhancer of the Col2a1 gene are required for cartilage-specific expression in vivo.

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