Induction of class I antigen processing components in oligodendroglia and microglia during viral encephalomyelitis. (Q30229156)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:18205173%20AND%20SRC:MED&resulttype=core&format=json

1 January 2020

title

Induction of class I antigen processing components in oligodendroglia and microglia during viral encephalomyelitis (English)

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1 January 2020

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5

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1 January 2020

4

Wendy Macklin

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1 January 2020

author name string

Karen E Malone

series ordinal

1

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:18205173%20AND%20SRC:MED&resulttype=core&format=json

1 January 2020

Stephen A Stohlman

series ordinal

2

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1 January 2020

Chandran Ramakrishna

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3

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1 January 2020

publication date

1 March 2008

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1 January 2020

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1 January 2020

volume

56

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1 January 2020

issue

4

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1 January 2020

page(s)

426-435

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Organization and functional analysis of the mouse transporter associated with antigen processing 2 promoter

1 January 2020

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Perforin-mediated effector function within the central nervous system requires IFN-gamma-mediated MHC up-regulation.

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Transcriptional regulation of MHC class I gene expression in rat oligodendrocytes.

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Vaccine-induced memory CD8+ T cells cannot prevent central nervous system virus reactivation

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In vivo expression of major histocompatibility complex molecules on oligodendrocytes and neurons during viral infection

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Myelin antigen-specific CD8+ T cells are encephalitogenic and produce severe disease in C57BL/6 mice

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Synergistic stimulation of MHC class I and IRF-1 gene expression by IFN-gamma and TNF-alpha in oligodendrocytes.

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Pathogenicity of antigenic variants of murine coronavirus JHM selected with monoclonal antibodies

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IFN-beta mediates coordinate expression of antigen-processing genes in RSV-infected pulmonary epithelial cells

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Purification and analysis of in vivo-differentiated oligodendrocytes expressing the green fluorescent protein

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Clonal expansions of CD8(+) T cells dominate the T cell infiltrate in active multiple sclerosis lesions as shown by micromanipulation and single cell polymerase chain reaction

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Haplotype matching is not an essential requirement to achieve remyelination of demyelinating CNS lesions.

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Multiple sclerosis: brain-infiltrating CD8+ T cells persist as clonal expansions in the cerebrospinal fluid and blood

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A pathogenic role for myelin-specific CD8(+) T cells in a model for multiple sclerosis

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Transcription of H-2 and Qa genes in embryonic and adult mice

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Inhibition of interferon-gamma signaling in oligodendroglia delays coronavirus clearance without altering demyelination

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Mouse hepatitis virus is cleared from the central nervous systems of mice lacking perforin-mediated cytolysis.

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Regulation of murine Tap1 and Lmp2 genes in macrophages by interferon gamma is mediated by STAT1 and IRF-1.

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Interferon-gamma, the functional plasticity of the ubiquitin-proteasome system, and MHC class I antigen processing

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Accessory molecules in the assembly of major histocompatibility complex class I/peptide complexes: how essential are they for CD8(+) T-cell immune responses?

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Type I interferons are essential in controlling neurotropic coronavirus infection irrespective of functional CD8 T cells.

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Major histocompatibility complex heavy chain accumulation in the endoplasmic reticulum of oligodendrocytes results in myelin abnormalities

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Interferon-gamma inhibits central nervous system remyelination through a process modulated by endoplasmic reticulum stress.

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Neurons produce type I interferon during viral encephalitis

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Evolutionary and functional perspectives of the major histocompatibility complex class I antigen-processing machinery

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IFN regulatory factor-1 plays a central role in the regulation of the expression of class I and II MHC genes in vivo.

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Major histocompatibility complex class I expression in oligodendrocytes induces hypomyelination in transgenic mice.

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Immune cells contribute to myelin degeneration and axonopathic changes in mice overexpressing proteolipid protein in oligodendrocytes

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Distinct regulation of MHC molecule expression on astrocytes and microglia during viral encephalomyelitis.

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Sequential infection of glial cells by the murine hepatitis virus JHM strain (MHV-4) leads to a characteristic distribution of demyelination

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Dependence of peptide binding by MHC class I molecules on their interaction with TAP.

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Coronavirus infection of the central nervous system: host-virus stand-off

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Dysmyelination in transgenic mice resulting from expression of class I histocompatibility molecules in oligodendrocytes.

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12 December 2020

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Selective loss of H-2Ds antigen on a murine B lymphoma due to a post-transcriptional block in expression

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12 December 2020

Major histocompatibility complex Class I expression in oligodendrocytes induces hypomyelination in transgenic mice

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https://api.crossref.org/works/10.1002%2FGLIA.20625

7 January 2021

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Identifiers

DOI

10.1002/GLIA.20625

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:18205173%20AND%20SRC:MED&resulttype=core&format=json

release_f4m7po6qfvav7pdxpcmbkmkt3q

1 January 2020

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Fatcat

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24 November 2022

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