Ebola virus and severe acute respiratory syndrome coronavirus display late cell entry kinetics: evidence that transport to NPC1+ endolysosomes is a rate-defining step (Q30301030)

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English	Ebola virus and severe acute respiratory syndrome coronavirus display late cell entry kinetics: evidence that transport to NPC1+ endolysosomes is a rate-defining step	scientific article

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scholarly article

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26 February 2020

Ebola virus and severe acute respiratory syndrome coronavirus display late cell entry kinetics: evidence that transport to NPC1+ endolysosomes is a rate-defining step (English)

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26 February 2020

Orthoebolavirus zairense

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inferred from title

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severe acute respiratory syndrome

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Charles J Shoemaker

3

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author name string

Rebecca M Mingo

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26 February 2020

James A Simmons

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26 February 2020

Elizabeth A Nelson

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26 February 2020

Kathryn L Schornberg

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26 February 2020

Ryan S D'Souza

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James E Casanova

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Judith M White

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publication date

31 December 2014

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26 February 2020

Journal of Virology

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26 February 2020

89

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26 February 2020

2931-2943

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26 February 2020

5

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26 February 2020

describes a project that uses

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Coronavirus cell entry occurs through the endo-/lysosomal pathway in a proteolysis-dependent manner.

1 reference

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Genomic surveillance elucidates Ebola virus origin and transmission during the 2014 outbreak

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Rotaviruses reach late endosomes and require the cation-dependent mannose-6-phosphate receptor and the activity of cathepsin proteases to enter the cell

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Chinese hamster ovary cell lines selected for resistance to ebolavirus glycoprotein mediated infection are defective for NPC1 expression.

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Simultaneous treatment of human bronchial epithelial cells with serine and cysteine protease inhibitors prevents severe acute respiratory syndrome coronavirus entry

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Cathepsins: fundamental effectors of endolysosomal proteolysis

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3 June 2018

Defective endocytic trafficking of NPC1 and NPC2 underlying infantile Niemann-Pick type C disease

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inferred from PubMed ID database lookup

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10.1128/JVI.03398-14

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26 February 2020

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26 February 2020

1 reference

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26 February 2020

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