Hypoxia death stimulus induces translocation of p53 protein to mitochondria. Detection by immunofluorescence on whole cells (Q31864081)

4 July 2017

Hypoxia death stimulus induces translocation of p53 protein to mitochondria. Detection by immunofluorescence on whole cells (English)

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immunofluorescence microscopy

4 July 2017

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Sansome C

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4 July 2017

Zaika A

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2

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4 July 2017

Marchenko ND

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3

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4 July 2017

Moll UM

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4

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4 July 2017

language of work or name

English

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publication date

1 January 2001

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488

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110-115

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p53, the cellular gatekeeper for growth and division

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Wild-type human p53 and a temperature-sensitive mutant induce Fas/APO-1 expression

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p53 activates the CD95 (APO-1/Fas) gene in response to DNA damage by anticancer drugs

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KILLER/DR5 is a DNA damage-inducible p53-regulated death receptor gene

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A novel form of Epstein-Barr virus latency in normal B cells in vivo

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Noxa, a BH3-only member of the Bcl-2 family and candidate mediator of p53-induced apoptosis

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p53AIP1, a potential mediator of p53-dependent apoptosis, and its regulation by Ser-46-phosphorylated p53

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A model for p53-induced apoptosis

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Involvement of p85 in p53-dependent apoptotic response to oxidative stress

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p53-dependent apoptosis in the absence of transcriptional activation of p53-target genes

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Myc-mediated apoptosis requires wild-type p53 in a manner independent of cell cycle arrest and the ability of p53 to induce p21waf1/cip1.

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Activation of caspases in p53-induced transactivation-independent apoptosis.

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Induction of apoptosis in HeLa cells by trans-activation-deficient p53.

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Cell surface trafficking of Fas: a rapid mechanism of p53-mediated apoptosis.

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Oncogene-dependent regulation of caspase activation by p53 protein in a cell-free system

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Smac, a mitochondrial protein that promotes cytochrome c-dependent caspase activation by eliminating IAP inhibition

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Identification of DIABLO, a mammalian protein that promotes apoptosis by binding to and antagonizing IAP proteins

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Molecular characterization of mitochondrial apoptosis-inducing factor

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The caspase-3 precursor has a cytosolic and mitochondrial distribution: implications for apoptotic signaling

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The central effectors of cell death in the immune system

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Mitochondria at the crossroad of apoptotic cell death

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Death signal-induced localization of p53 protein to mitochondria. A potential role in apoptotic signaling

21 January 2018

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Stress signals utilize multiple pathways to stabilize p53

21 January 2018

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Hypoxia induces accumulation of p53 protein, but activation of a G1-phase checkpoint by low-oxygen conditions is independent of p53 status.

21 January 2018

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Stabilization of wild-type p53 by hypoxia-inducible factor 1alpha

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Cobalt and desferrioxamine reveal crucial members of the oxygen sensing pathway in HepG2 cells

21 January 2018

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Reactive oxygen species generated at mitochondrial complex III stabilize hypoxia-inducible factor-1alpha during hypoxia: a mechanism of O2 sensing

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Cytochrome c release and apoptosis induced by mitochondrial targeting of nuclear orphan receptor TR3.

21 January 2018

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p53 levels, functional domains, and DNA damage determine the extent of the apoptotic response of tumor cells

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Specific loss of apoptotic but not cell-cycle arrest function in a human tumor derived p53 mutant

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DNA damage induces p21 protein expression by inhibiting ubiquitination in ML-1 cells

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Iron deprivation-induced apoptosis in HL-60 cells

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Hypoxia-induced vascular endothelial growth factor expression in normal rat astrocyte cultures

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Translocation of apocytochrome c across the outer membrane of mitochondria

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Apoptosis. Mitochondria--the death signal integrators

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10.1016/S0014-5793(00)02368-1

7 January 2021

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DOI

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4 July 2017

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