Activators of AMP-activated protein kinase enhance GLUT4 translocation and its glucose transport activity in 3T3-L1 adipocytes (Q33216242)

24 July 2017

Activators of AMP-activated protein kinase enhance GLUT4 translocation and its glucose transport activity in 3T3-L1 adipocytes (English)

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24 July 2017

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Shinya Yamaguchi

1

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24 July 2017

Hiroshi Katahira

2

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24 July 2017

Sachihiko Ozawa

3

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24 July 2017

Yoko Nakamichi

4

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24 July 2017

Toshiaki Tanaka

5

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24 July 2017

Tatsuhiro Shimoyama

6

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24 July 2017

Kazuto Takahashi

7

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24 July 2017

Katsuhiko Yoshimoto

8

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24 July 2017

Mica Ohara Imaizumi

9

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24 July 2017

Shinya Nagamatsu

10

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24 July 2017

Hitoshi Ishida

11

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24 July 2017

31 May 2005

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American Journal of Physiology - Endocrinology and Metabolism

24 July 2017

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24 July 2017

289

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24 July 2017

E643-9

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24 July 2017

4

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Stimulation of AMP-activated protein kinase (AMPK) is associated with enhancement of Glut1-mediated glucose transport

24 July 2017

1 reference

https://api.crossref.org/works/10.1152%2FAJPENDO.00456.2004

Hyperosmotic shock induces both activation and translocation of glucose transporters in mammalian cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPENDO.00456.2004

Arsenite stimulated glucose transport in 3T3-L1 adipocytes involves both Glut4 translocation and p38 MAPK activity.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPENDO.00456.2004

Osmotic shock stimulates GLUT4 translocation in 3T3L1 adipocytes by a novel tyrosine kinase pathway

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPENDO.00456.2004

Activation of the ERK pathway and atypical protein kinase C isoforms in exercise- and aminoimidazole-4-carboxamide-1-beta-D-riboside (AICAR)-stimulated glucose transport

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPENDO.00456.2004

5-aminoimidazole-4-carboxamide ribonucleoside. A specific method for activating AMP-activated protein kinase in intact cells?

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPENDO.00456.2004

Differential effects of constitutively active phosphatidylinositol 3-kinase on glucose transport, glycogen synthase activity, and DNA synthesis in 3T3-L1 adipocytes

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPENDO.00456.2004

Protein kinase inhibitors block the stimulation of the AMP-activated protein kinase by 5-amino-4-imidazolecarboxamide riboside.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPENDO.00456.2004

The AMP-activated protein kinase--fuel gauge of the mammalian cell?

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPENDO.00456.2004

Sequence, tissue distribution, and differential expression of mRNA for a putative insulin-responsive glucose transporter in mouse 3T3-L1 adipocytes

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPENDO.00456.2004

Insulin-stimulated GLUT4 translocation is relevant to the phosphorylation of IRS-1 and the activity of PI3-kinase

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPENDO.00456.2004

Overexpression of catalytic subunit p110alpha of phosphatidylinositol 3-kinase increases glucose transport activity with translocation of glucose transporters in 3T3-L1 adipocytes

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPENDO.00456.2004

Bradykinin directly triggers GLUT4 translocation via an insulin-independent pathway.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPENDO.00456.2004

Requirement for phosphoinositide 3-kinase in insulin-stimulated GLUT4 translocation in 3T3-L1 adipocytes

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPENDO.00456.2004

5' AMP-activated protein kinase activation causes GLUT4 translocation in skeletal muscle

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPENDO.00456.2004

The AMP-activated protein kinase activator AICAR does not induce GLUT4 translocation to transverse tubules but stimulates glucose uptake and p38 mitogen-activated protein kinases alpha and beta in skeletal muscle

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPENDO.00456.2004

A role for AMP-activated protein kinase in contraction- and hypoxia-regulated glucose transport in skeletal muscle

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPENDO.00456.2004

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPENDO.00456.2004

GLUT4 vesicle dynamics in living 3T3 L1 adipocytes visualized with green-fluorescent protein

21 January 2018

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https://api.crossref.org/works/10.1152%2FAJPENDO.00456.2004

Insulin action on GLUT4 traffic visualized in single 3T3-l1 adipocytes by using ultra-fast microscopy

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPENDO.00456.2004

Activation of AMPK is essential for AICAR-induced glucose uptake by skeletal muscle but not adipocytes

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPENDO.00456.2004

5-aminoimidazole-4-carboxamide ribonucleoside (AICAR) inhibits insulin-stimulated glucose transport in 3T3-L1 adipocytes.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPENDO.00456.2004

Differential effects of phosphatidylinositol 3-kinase inhibition on intracellular signals regulating GLUT4 translocation and glucose transport

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPENDO.00456.2004

A dominant-negative p38 MAPK mutant and novel selective inhibitors of p38 MAPK reduce insulin-stimulated glucose uptake in 3T3-L1 adipocytes without affecting GLUT4 translocation

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPENDO.00456.2004

Stimulation of glucose transport by AMP-activated protein kinase via activation of p38 mitogen-activated protein kinase

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPENDO.00456.2004

10.1152/AJPENDO.00456.2004

21 January 2018

Identifiers

DOI

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24 July 2017

1 reference