The NF-kappaB pathway blockade by the IKK inhibitor PS1145 can overcome imatinib resistance (Q33226589)

24 July 2017

The NF-kappaB pathway blockade by the IKK inhibitor PS1145 can overcome imatinib resistance (English)

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24 July 2017

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Cilloni D

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Messa F

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Arruga F

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Defilippi I

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Morotti A

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Messa E

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Carturan S

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Giugliano E

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Pautasso M

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Bracco E

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Rosso V

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Sen A

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Martinelli G

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Baccarani M

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Saglio G

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1 January 2006

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61-67

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https://scigraph.springernature.com/pub.10.1038/sj.leu.2403998

24 July 2017

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Hematologic and cytogenetic responses to imatinib mesylate in chronic myelogenous leukemia

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Imatinib compared with interferon and low-dose cytarabine for newly diagnosed chronic-phase chronic myeloid leukemia

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Imatinib mesylate therapy in newly diagnosed patients with Philadelphia chromosome-positive chronic myelogenous leukemia: high incidence of early complete and major cytogenetic responses

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Imatinib mesylate (STI571) therapy for Philadelphia chromosome-positive chronic myelogenous leukemia in blast phase

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Activity of a specific inhibitor of the BCR-ABL tyrosine kinase in the blast crisis of chronic myeloid leukemia and acute lymphoblastic leukemia with the Philadelphia chromosome

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Molecular mechanisms of resistance to imatinib in Philadelphia-chromosome-positive leukaemias.

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Clinical resistance to STI-571 cancer therapy caused by BCR-ABL gene mutation or amplification

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Detection of BCR-ABL mutations in patients with CML treated with imatinib is virtually always accompanied by clinical resistance, and mutations in the ATP phosphate-binding loop (P-loop) are associated with a poor prognosis

21 January 2018

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Missing pieces in the NF-kappaB puzzle

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NF-kappaB in cancer: a marked target

21 January 2018

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Nuclear transcription factor-kappaB as a target for cancer drug development

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Nuclear factor-kappaB is constitutively activated in primitive human acute myelogenous leukemia cells

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Expression of constitutively active nuclear-kappa B RelA transcription factor in blasts of acute myeloid leukemia

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Transcription factor NF-kappaB is constitutively activated in acute lymphoblastic leukemia cells.

21 January 2018

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Nuclear factor-kappaB modulation as a therapeutic approach in hematologic malignancies

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Phosphorylation meets ubiquitination: the control of NF-[kappa]B activity

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NF-kappaB at the crossroads of life and death

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The IKK/NF-kappa B pathway

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NF-kappa B as a therapeutic target in multiple myeloma

21 January 2018

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NF-kappaB signaling and human disease

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Activators and target genes of Rel/NF-kappaB transcription factors

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The IKKbeta subunit of IkappaB kinase (IKK) is essential for nuclear factor kappaB activation and prevention of apoptosis

21 January 2018

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Direct Phosphorylation of IB by IKK and IKK: Discrimination Between Free and NF-B-Bound Substrate

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Positive and negative regulation of IkappaB kinase activity through IKKbeta subunit phosphorylation

21 January 2018

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Inactivation of NF-kappaB-dependent cell survival, a novel mechanism for the proapoptotic function of c-Abl

21 January 2018

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c-Abl: activation and nuclear targets

21 January 2018

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The Abl family kinases: mechanisms of regulation and signaling

21 January 2018

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A requirement for NF-kappaB activation in Bcr-Abl-mediated transformation

21 January 2018

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Increased IkappaB kinase activity is associated with activated NF-kappaB in acute myeloid blasts

21 January 2018

1 reference

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inferred from DOI database lookup

7 January 2021

based on heuristic

Natural resistance of acute myeloid leukemia cell lines to mitoxantrone is associated with lack of apoptosis

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inferred from DOI database lookup

7 January 2021

based on heuristic

Novel therapies for multiple myeloma

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2403998

inferred from DOI database lookup

7 January 2021

based on heuristic

Identifiers

DOI

10.1038/SJ.LEU.2403998

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Dimensions Publication ID

24 July 2017

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