Aberrant epigenetic silencing is triggered by a transient reduction in gene expression (Q33417274)

26 July 2017

Aberrant epigenetic silencing is triggered by a transient reduction in gene expression (English)

1 reference

26 July 2017

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Jon A Oyer

1

1 reference

26 July 2017

Adrian Chu

series ordinal

2

1 reference

26 July 2017

Sukhmani Brar

series ordinal

3

1 reference

26 July 2017

Mitchell S Turker

series ordinal

4

1 reference

26 July 2017

language of work or name

English

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publication date

12 March 2009

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26 July 2017

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26 July 2017

4

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26 July 2017

3

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26 July 2017

e4832

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Creative Commons Attribution 4.0 International

26 July 2017

start time

12 March 2009

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April 2022 Public Data File from Crossref

copyright status

copyrighted

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cites work

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Histone H3 acetylation and H3 K4 methylation define distinct chromatin regions permissive for transgene expression

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Nickel ions increase histone H3 lysine 9 dimethylation and induce transgene silencing

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Silenced tumor suppressor genes reactivated by DNA demethylation do not return to a fully euchromatic chromatin state

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Hypoxia-induced down-regulation of BRCA1 expression by E2Fs.

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Chromatin inactivation precedes de novo DNA methylation during the progressive epigenetic silencing of the RASSF1A promoter

21 June 2018

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Histone methyltransferases G9a and GLP form heteromeric complexes and are both crucial for methylation of euchromatin at H3-K9

21 June 2018

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Loss of estrogen receptor signaling triggers epigenetic silencing of downstream targets in breast cancer

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Transcriptional silencing of the RUNX3 gene by CpG hypermethylation is associated with lung cancer.

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Silencing of mouse Aprt is a gradual process in differentiated cells

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Histone modifications and silencing prior to DNA methylation of a tumor suppressor gene.

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Dependence of histone modifications and gene expression on DNA hypermethylation in cancer.

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Tandem B1 elements located in a mouse methylation center provide a target for de novo DNA methylation

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12 December 2020

Methylation-independent silencing of the p73 gene in neuroblastoma

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The VP16 activation domain interacts with multiple transcriptional components as determined by protein-protein cross-linking in vivo

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Methylation-independent silencing of the tumor suppressor INK4b (p15) by CBFbeta-SMMHC in acute myelogenous leukemia with inv(16)

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10.1371/JOURNAL.PONE.0004832

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DOI

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26 July 2017

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26 July 2017

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