Phage-derived fully human monoclonal antibody fragments to human vascular endothelial growth factor-C block its interaction with VEGF receptor-2 and 3. (Q33651167)

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English	Phage-derived fully human monoclonal antibody fragments to human vascular endothelial growth factor-C block its interaction with VEGF receptor-2 and 3.	scientific article

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scholarly article

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Europe PubMed Central

PubMed publication ID

28 July 2017

Phage-derived fully human monoclonal antibody fragments to human vascular endothelial growth factor-C block its interaction with VEGF receptor-2 and 3. (English)

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Europe PubMed Central

PubMed publication ID

28 July 2017

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monoclonal antibody

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based on heuristic

inferred from title

4

object named as

Dario Neri

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Europe PubMed Central

PubMed publication ID

28 July 2017

5

object named as

Michael Detmar

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Europe PubMed Central

PubMed publication ID

28 July 2017

author name string

Matthias Rinderknecht

1

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Europe PubMed Central

PubMed publication ID

28 July 2017

Alessandra Villa

2

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Europe PubMed Central

PubMed publication ID

28 July 2017

Kurt Ballmer-Hofer

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Europe PubMed Central

PubMed publication ID

28 July 2017

language of work or name

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publication date

2 August 2010

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Europe PubMed Central

PubMed publication ID

28 July 2017

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Europe PubMed Central

PubMed publication ID

28 July 2017

5

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Europe PubMed Central

PubMed publication ID

28 July 2017

8

1 reference

Europe PubMed Central

PubMed publication ID

28 July 2017

e11941

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Europe PubMed Central

PubMed publication ID

28 July 2017

copyright license

Creative Commons Attribution 4.0 International

2 August 2010

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April 2022 Public Data File from Crossref

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Structural determinants of growth factor binding and specificity by VEGF receptor 2

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Autocrine loop between vascular endothelial growth factor (VEGF)-C and VEGF receptor-3 positively regulates tumor-associated lymphangiogenesis in oral squamoid cancer cells

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Alternatively spliced vascular endothelial growth factor receptor-2 is an essential endogenous inhibitor of lymphatic vessel growth.

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Vascular endothelial growth factor C stimulates progression of human gastric cancer via both autocrine and paracrine mechanisms.

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VEGF-C mediates cyclic pressure-induced endothelial cell proliferation

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The role of tumor lymphangiogenesis in metastatic spread.

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Suppression of tumor lymphangiogenesis and lymph node metastasis by blocking vascular endothelial growth factor receptor 3 signaling.

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Vascular endothelial growth factor (VEGF)-C signaling through FLT-4 (VEGFR-3) mediates leukemic cell proliferation, survival, and resistance to chemotherapy

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Identifiers

10.1371/JOURNAL.PONE.0011941

1 reference

Europe PubMed Central

PubMed publication ID

28 July 2017

2010PLoSO...511941R

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PMC publication ID

1 reference

Europe PubMed Central

PubMed publication ID

28 July 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

28 July 2017

ResearchGate publication ID

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