Transcription profiling of Epstein-Barr virus nuclear antigen (EBNA)-1 expressing cells suggests targeting of chromatin remodeling complexes (Q33656437)

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English	Transcription profiling of Epstein-Barr virus nuclear antigen (EBNA)-1 expressing cells suggests targeting of chromatin remodeling complexes	scientific article

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scholarly article

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Europe PubMed Central

28 July 2017

Transcription profiling of Epstein-Barr virus nuclear antigen (EBNA)-1 expressing cells suggests targeting of chromatin remodeling complexes (English)

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Europe PubMed Central

28 July 2017

Epstein–Barr virus

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Maria G Masucci

4

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Ramakrishna Sompallae

1

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Europe PubMed Central

28 July 2017

Simone Callegari

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Europe PubMed Central

28 July 2017

Siamak Akbari Kamranvar

3

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Europe PubMed Central

28 July 2017

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10 August 2010

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Europe PubMed Central

28 July 2017

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Europe PubMed Central

28 July 2017

5

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Europe PubMed Central

28 July 2017

8

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Europe PubMed Central

28 July 2017

e12052

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Europe PubMed Central

28 July 2017

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Creative Commons Attribution 4.0 International

10 August 2010

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April 2022 Public Data File from Crossref

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Cytoscape

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Europe PubMed Central

9 June 2022

https://europepmc.org/search?query=(METHODS:'Cytoscape')

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CZ Software Mentions: A large dataset of software mentions in the biomedical literature

EBNA1-mediated recruitment of a histone H2B deubiquitylating complex to the Epstein-Barr virus latent origin of DNA replication

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Histone levels are regulated by phosphorylation and ubiquitylation-dependent proteolysis

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The biology of chromatin remodeling complexes

1 reference

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The Epstein-Barr virus nuclear antigen-1 promotes genomic instability via induction of reactive oxygen species.

1 reference

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Identifying sites bound by Epstein-Barr virus nuclear antigen 1 (EBNA1) in the human genome: defining a position-weighted matrix to predict sites bound by EBNA1 in viral genomes

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Epstein-Barr virus-encoded EBNA1 modulates the AP-1 transcription factor pathway in nasopharyngeal carcinoma cells and enhances angiogenesis in vitro

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Histone ubiquitination: triggering gene activity

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The amino terminus of Epstein-Barr Virus (EBV) nuclear antigen 1 contains AT hooks that facilitate the replication and partitioning of latent EBV genomes by tethering them to cellular chromosomes

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1 reference

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1 reference

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An EBNA-1-dependent enhancer acts from a distance of 10 kilobase pairs to increase expression of the Epstein-Barr virus LMP gene

1 reference

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trans activation of an Epstein-Barr viral transcriptional enhancer by the Epstein-Barr viral nuclear antigen 1.

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2919392

21 June 2018

Sequence-specific DNA binding of the Epstein-Barr virus nuclear antigen (EBNA-1) to clustered sites in the plasmid maintenance region

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2919392

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Multiple EBNA1-binding sites are required to form an EBNA1-dependent enhancer and to activate a minimal replicative origin within oriP of Epstein-Barr virus.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2919392

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A promoter of Epstein-Barr virus that can function during latent infection can be transactivated by EBNA-1, a viral protein required for viral DNA replication during latent infection.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2919392

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The Epstein-Barr virus nuclear protein 1 promoter active in type I latency is autoregulated

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Evidence for a novel gene associated with low tumor metastatic potential

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https://pubmed.ncbi.nlm.nih.gov/20706582

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Novel hypotheses for the roles of EBNA-1 and BHRF1 in EBV-related cancers

1 reference

https://pubmed.ncbi.nlm.nih.gov/20706582

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1 reference

https://pubmed.ncbi.nlm.nih.gov/20706582

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based on heuristic

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10.1371/JOURNAL.PONE.0012052

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Europe PubMed Central

28 July 2017

2010PLoSO...512052S

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Europe PubMed Central

28 July 2017

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Europe PubMed Central

28 July 2017

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