Immune responses to adenoviruses: viral evasion mechanisms and their implications for the clinic. (Q33711437)

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11 December 2019

review article

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title

Immune responses to adenoviruses: viral evasion mechanisms and their implications for the clinic (English)

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11 December 2019

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3

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11 December 2019

author name string

Wold WS

1

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11 December 2019

Doronin K

2

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11 December 2019

Kuppuswamy M

4

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11 December 2019

Lichtenstein DL

5

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11 December 2019

Tollefson AE

6

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11 December 2019

publication date

1 August 1999

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Current Opinion in Immunology

11 December 2019

published in

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11 December 2019

volume

11

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11 December 2019

issue

4

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11 December 2019

page(s)

380-386

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Impact of preexisting and induced humoral and cellular immune responses in an adenovirus-based gene therapy phase I clinical trial for localized mesothelioma.

11 December 2019

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Death receptor 5, a new member of the TNFR family, and DR4 induce FADD-dependent apoptosis and activate the NF-kappaB pathway

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The CED-4-homologous protein FLASH is involved in Fas-mediated activation of caspase-8 during apoptosis

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TNF-dependent recruitment of the protein kinase RIP to the TNF receptor-1 signaling complex

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The death domain kinase RIP mediates the TNF-induced NF-kappaB signal

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Forced degradation of Fas inhibits apoptosis in adenovirus-infected cells

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The adenovirus E3-10.4K/14.5K complex mediates loss of cell surface Fas (CD95) and resistance to Fas-induced apoptosis

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The 10,400- and 14,500-dalton proteins encoded by region E3 of adenovirus form a complex and function together to down-regulate the epidermal growth factor receptor

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The medium subunits of adaptor complexes recognize distinct but overlapping sets of tyrosine-based sorting signals

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Adenovirus E3-10.4K/14.5K protein complex inhibits tumor necrosis factor-induced translocation of cytosolic phospholipase A2 to membranes

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Properties and regulation of cytosolic phospholipase A2.

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The adenovirus E3-14.7K protein and the E3-10.4K/14.5K complex of proteins, which independently inhibit tumor necrosis factor (TNF)-induced apoptosis, also independently inhibit TNF-induced release of arachidonic acid

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7 January 2021

A 14,700 MW protein from the E3 region of adenovirus inhibits cytolysis by tumor necrosis factor

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7 January 2021

Interaction of the adenovirus 14.7-kDa protein with FLICE inhibits Fas ligand-induced apoptosis

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7 January 2021

Interaction of an adenovirus E3 14.7-kilodalton protein with a novel tumor necrosis factor alpha-inducible cellular protein containing leucine zipper domains

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7 January 2021

E1B 19K inhibits Fas-mediated apoptosis through FADD-dependent sequestration of FLICE.

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7 January 2021

Lung-specific expression of adenovirus E3-14.7K in transgenic mice attenuates adenoviral vector-mediated lung inflammation and enhances transgene expression

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Fas-Fas ligand interactions play a major role in effector functions of cytotoxic T lymphocytes after adenovirus vector-mediated gene transfer

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Expression of gp19K increases the persistence of transgene expression from an adenovirus vector in the mouse lung and liver

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7 January 2021

10.1016/S0952-7915(99)80064-8

Identifiers

DOI

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10448144%20AND%20SRC:MED&resulttype=core&format=json

11 December 2019

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