Neurofibrillary degeneration of the Alzheimer-type: an alternate pathway to neuronal apoptosis? (Q33973135)

30 July 2017

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title

Neurofibrillary degeneration of the Alzheimer-type: an alternate pathway to neuronal apoptosis? (English)

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30 July 2017

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Malika Hamdane

1

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Caroline Smet

Caroline Smet

4

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Guy Lippens

Guy Lippens

9

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Luc Buée

Luc Buée

11

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Isabelle Landrieu

7

Isabelle Landrieu

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30 July 2017

3

Anne-Véronique Sambo

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30 July 2017

Patrice Delobel

2

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30 July 2017

Séverine Bégard

5

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30 July 2017

Anne Violleau

6

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30 July 2017

André Delacourte

8

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30 July 2017

Stéphane Flament

10

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30 July 2017

language of work or name

English

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publication date

1 October 2003

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30 July 2017

7

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based on heuristic

inferred from page(s)

published in

Biochemical Pharmacology

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30 July 2017

66

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30 July 2017

8

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30 July 2017

1619-1625

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The amyloid hypothesis of Alzheimer's disease: progress and problems on the road to therapeutics

30 July 2017

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https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Tau protein isoforms, phosphorylation and role in neurodegenerative disorders11These authors contributed equally to this work

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Nonoverlapping but synergetic tau and APP pathologies in sporadic Alzheimer's disease.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Tau blocks traffic of organelles, neurofilaments, and APP vesicles in neurons and enhances oxidative stress

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Alzheimer's disease: a dysfunction of the amyloid precursor protein(1).

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Immunohistochemical evidence for apoptosis in Alzheimer's disease.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Par-4 is a mediator of neuronal degeneration associated with the pathogenesis of Alzheimer disease.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Relationship between DNA fragmentation, morphological changes and neuronal loss in Alzheimer's disease and dementia with Lewy bodies.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Alzheimer disease: DNA fragmentation indicates increased neuronal vulnerability, but not apoptosis.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Research communication copper-1,10-phenanthroline induces internucleosomal DNA fragmentation in HepG2 cells, resulting from direct oxidation by the hydroxyl radical

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Analysis of cell cycle-related gene expression in postmitotic neurons: selective induction of Cyclin D1 during programmed cell death

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Role of cell cycle regulatory proteins in cerebellar granule neuron apoptosis.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Neuronal apoptosis at the G1/S cell cycle checkpoint

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Mild cerebral ischemia induces loss of cyclin-dependent kinase inhibitors and activation of cell cycle machinery before delayed neuronal cell death.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Neuronal expression of cycline dependent kinase inhibitors of the INK4 family in Alzheimer's disease.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Proline-directed phosphorylation and isomerization in mitotic regulation and in Alzheimer's Disease

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Cell cycle regulatory failure in neurones: causes and consequences.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Neurons in Alzheimer disease emerge from senescence.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Aberrant expression of mitotic cdc2/cyclin B1 kinase in degenerating neurons of Alzheimer's disease brain.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Mitotic phosphoepitopes precede paired helical filaments in Alzheimer's disease

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Tau proteins of Alzheimer paired helical filaments: abnormal phosphorylation of all six brain isoforms

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Neurofibrillary tangles and tau phosphorylation.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Modelling Alzheimer-specific abnormal Tau phosphorylation independently of GSK3beta and PKA kinase activities.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Sequential phosphorylation of Tau by glycogen synthase kinase-3beta and protein kinase A at Thr212 and Ser214 generates the Alzheimer-specific epitope of antibody AT100 and requires a paired-helical-filament-like conformation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Phosphorylation of microtubule-associated protein tau by stress-activated protein kinases in intact cells

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Induction of a specific tau Alzheimer epitope in SY-5Y neuroblastoma cells.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Pathological tau phenotypes. The weight of mutations, polymorphisms, and differential neuronal vulnerabilities.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Mitotic mechanisms in Alzheimer's disease?

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

A conformation- and phosphorylation-dependent antibody recognizing the paired helical filaments of Alzheimer's disease.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

The prolyl isomerase Pin1 restores the function of Alzheimer-associated phosphorylated tau protein

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Mitotic activation: a convergent mechanism for a cohort of neurodegenerative diseases.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Abnormal Tau phosphorylation of the Alzheimer-type also occurs during mitosis.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

1H NMR study on the binding of Pin1 Trp-Trp domain with phosphothreonine peptides

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Specific tau phosphorylation sites correlate with severity of neuronal cytopathology in Alzheimer's disease.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Conversion of p35 to p25 deregulates Cdk5 activity and promotes neurodegeneration

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Deregulation of cdk5, hyperphosphorylation, and cytoskeletal pathology in the Niemann-Pick type C murine model.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Neuron-specific phosphorylation of Alzheimer's beta-amyloid precursor protein by cyclin-dependent kinase 5.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Neurotoxicity induces cleavage of p35 to p25 by calpain

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Pin1: a therapeutic target in Alzheimer neurodegeneration

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Critical role of WW domain phosphorylation in regulating phosphoserine binding activity and Pin1 function

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Inverse association of Pin1 and tau accumulation in Alzheimer's disease hippocampus

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Pin1 regulates turnover and subcellular localization of beta-catenin by inhibiting its interaction with APC

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Pin1 is overexpressed in breast cancer and cooperates with Ras signaling in increasing the transcriptional activity of c-Jun towards cyclin D1.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

IGF-1 induces Pin1 expression in promoting cell cycle S-phase entry

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

CDK inhibitors: positive and negative regulators of G1-phase progression

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Prolyl isomerase Pin1: a catalyst for oncogenesis and a potential therapeutic target in cancer

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

Cyclin-dependent kinase 5 (Cdk5) and neuron-specific Cdk5 activators

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-2952%2803%2900533-1

inferred from DOI database lookup

7 January 2021

based on heuristic

Identifiers

DOI

10.1016/S0006-2952(03)00533-1

1 reference

30 July 2017

1 reference