The redox enzyme p66Shc contributes to diabetes and ischemia-induced delay in cutaneous wound healing. (Q34082896)

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6 February 2020

title

The redox enzyme p66Shc contributes to diabetes and ischemia-induced delay in cutaneous wound healing (English)

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6 February 2020

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1

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Mattia Albiero

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6 February 2020

Vittorio Pengo

9

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Carlo Agostini

11

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6 February 2020

Marco Giorgio

13

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14

Angelo Avogaro

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3

Lisa Menegazzo

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Elisa Pagnin

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8

Annunziata Lapolla

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6 February 2020

6

Elisabetta Iori

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6 February 2020

author name string

Elisa Boscaro

4

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6 February 2020

Chiara Cosma

7

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6 February 2020

Massimo Stendardo

10

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Pier Giuseppe Pelicci

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language of work or name

English

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publication date

21 June 2010

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6 February 2020

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6 February 2020

volume

59

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6 February 2020

page(s)

2306-2314

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6 February 2020

issue

9

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Creative Commons Attribution-NonCommercial-NoDerivs 3.0 Unported

6 February 2020

copyright license

start time

27 August 2021

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April 2022 Public Data File from Crossref

copyright status

copyrighted

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cites work

Decreased superoxide production in macrophages of long-lived p66Shc knock-out mice.

1 reference

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The molecular basis for impaired hypoxia-induced VEGF expression in diabetic tissues

5 July 2018

1 reference

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Apoptosis and oxidative stress-related diseases: the p66Shc connection

5 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2927954

Long-term prognosis of diabetic patients with critical limb ischemia: a population-based cohort study

5 July 2018

1 reference

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p66Shc, oxidative stress and aging: importing a lifespan determinant into mitochondria

5 July 2018

1 reference

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Genetic deletion of p66(Shc) adaptor protein prevents hyperglycemia-induced endothelial dysfunction and oxidative stress.

5 July 2018

1 reference

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Significance of endothelial progenitor cells in subjects with diabetes.

5 July 2018

1 reference

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The molecular biology of chronic wounds and delayed healing in diabetes

5 July 2018

1 reference

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The global burden of diabetic foot disease

5 July 2018

1 reference

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Molecular pathogenesis of chronic wounds: the role of beta-catenin and c-myc in the inhibition of epithelialization and wound healing

5 July 2018

1 reference

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The pathobiology of diabetic complications: a unifying mechanism

5 July 2018

1 reference

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Reducing the incidence of foot ulceration and amputation in diabetes

5 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2927954

Diabetes and atherosclerosis: epidemiology, pathophysiology, and management

5 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2927954

Cutaneous wound healing: an update

5 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2927954

The p66shc adaptor protein controls oxidative stress response and life span in mammals

5 July 2018

1 reference

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Inhibited proliferation of fibroblasts derived from chronic diabetic wounds and normal dermal fibroblasts treated with high glucose is associated with increased formation of l-lactate.

5 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2927954

p66ShcA modulates oxidative stress and survival of endothelial progenitor cells in response to high glucose

5 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2927954

Complexity of factors related to outcome of neuropathic and neuroischaemic/ischaemic diabetic foot ulcers: a cohort study

28 October 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2927954

28 October 2018

1 reference

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Diabetes induces p66shc gene expression in human peripheral blood mononuclear cells: relationship to oxidative stress.

28 October 2018

1 reference

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Monocyte NADPH oxidase subunit p22(phox) and inducible hemeoxygenase-1 gene expressions are increased in type II diabetic patients: relationship with oxidative stress

28 October 2018

1 reference

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Incidence, outcomes, and cost of foot ulcers in patients with diabetes.

28 October 2018

1 reference

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Electron Transfer between Cytochrome c and p66Shc Generates Reactive Oxygen Species that Trigger Mitochondrial Apoptosis

28 October 2018

1 reference

12 December 2020

Deletion of p66Shc longevity gene protects against experimental diabetic glomerulopathy by preventing diabetes-induced oxidative stress

1 reference

12 December 2020

Diabetes Promotes Cardiac Stem Cell Aging and Heart Failure, Which Are Prevented by Deletion of the p66 shc Gene

1 reference

12 December 2020

Diabetes impairs progenitor cell mobilisation after hindlimb ischaemia–reperfusion injury in rats

1 reference

12 December 2020

Identifiers

DOI

10.2337/DB09-1727

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:20566667%20AND%20SRC:MED&resulttype=core&format=json

6 February 2020

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:20566667%20AND%20SRC:MED&resulttype=core&format=json

6 February 2020

1 reference