Creation of psoriatic plaques: the ultimate tumor suppressor pathway. A new model for an ancient T-cell-mediated skin disease. Viewpoint (Q34135765)

31 July 2017

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title

Creation of psoriatic plaques: the ultimate tumor suppressor pathway. A new model for an ancient T-cell-mediated skin disease. Viewpoint (English)

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31 July 2017

1

Nickoloff BJ

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31 July 2017

1 February 2001

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Journal of Cutaneous Pathology

31 July 2017

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31 July 2017

28

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31 July 2017

2

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31 July 2017

57-64

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Psoriasis: a T-cell-mediated autoimmune disease induced by streptococcal superantigens?

31 July 2017

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Observations on mitosis in psoriatic epidermis

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Psoriasis: maintenance of an intact monolayer basal cell differentiation compartment in spite of hyperproliferation

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Treatment of psoriasis

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Apoptosis in proliferating, senescent, and immortalized keratinocytes

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The biology of replicative senescence

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Oncogenic ras provokes premature cell senescence associated with accumulation of p53 and p16INK4a

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The cat and mouse games that genes, viruses, and cells play

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Involvement of the cyclin-dependent kinase inhibitor p16 (INK4a) in replicative senescence of normal human fibroblasts

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Regulation of p16CDKN2 expression and its implications for cell immortalization and senescence

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Growth factor and proto-oncogene expression in psoriasis

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Evidence for an Alternative Pathway of Keratinocyte Maturation in Psoriasis from an Antigen Found in Psoriatic but Not Normal Epidermis

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Malignant transformation of mouse primary keratinocytes by Harvey sarcoma virus and its modulation by surrounding normal cells

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Escape from transforming growth factor beta control and oncogene cooperation in skin tumor development

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Defects in transforming growth factor-beta signaling cooperate with a Ras oncogene to cause rapid aneuploidy and malignant transformation of mouse keratinocytes

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Cell interactions control the fate of malignant keratinocytes in an organotypic model of early neoplasia

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Transforming growth factor-?1 localization in normal and psoriatic epidermal keratinocytes in situ

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Telomerase is not an epidermal stem cell marker and is downregulated by calcium

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Integration of positive and negative growth signals during ras pathway activation in vivo

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Repression of c-fos transcription and an altered genetic program in senescent human fibroblasts

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Autoradiographic analysis of turnover times of normal and psoriatic epidermis

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On cell kinetics in psoriasis

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Inhibitory function of p21Cip1/WAF1 in differentiation of primary mouse keratinocytes independent of cell cycle control

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Multiple stages and genetic alterations in immortalization, malignant transformation, and tumor progression of human skin keratinocytes

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Identifiers

DOI

1 reference

31 July 2017

1 reference

PubMed ID

11168753