Human papillomavirus type 16 E6 amino acid 83 variants enhance E6-mediated MAPK signaling and differentially regulate tumorigenesis by notch signaling and oncogenic Ras. (Q34150408)

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English	Human papillomavirus type 16 E6 amino acid 83 variants enhance E6-mediated MAPK signaling and differentially regulate tumorigenesis by notch signaling and oncogenic Ras.	scientific article

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28 December 2019

Human papillomavirus type 16 E6 amino acid 83 variants enhance E6-mediated MAPK signaling and differentially regulate tumorigenesis by notch signaling and oncogenic Ras (English)

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28 December 2019

Elliot J. Androphy

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28 December 2019

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Oishee Chakrabarti

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28 December 2019

Karthikeyan Veeraraghavalu

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28 December 2019

Vinay Tergaonkar

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28 December 2019

Yun Liu

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28 December 2019

Margaret A Stanley

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28 December 2019

Sudhir Krishna

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1 June 2004

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28 December 2019

Journal of Virology

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28 December 2019

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28 December 2019

5934-5945

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28 December 2019

Papillomavirus-mediated neoplastic progression is associated with reciprocal changes in JAGGED1 and manic fringe expression linked to notch activation

1 reference

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5 July 2018

Notch1 can contribute to viral-induced transformation of primary human keratinocytes

1 reference

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5 July 2018

Inhibition of the PI3K-Akt signaling pathway enhances the sensitivity of Fas-mediated apoptosis in human gastric carcinoma cell line, MKN-45

1 reference

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5 July 2018

Activated Notch1 inhibits p53-induced apoptosis and sustains transformation by human papillomavirus type 16 E6 and E7 oncogenes through a PI3K-PKB/Akt-dependent pathway

1 reference

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5 July 2018

The high-risk human papillomavirus type 16 E6 counters the GAP function of E6TP1 toward small Rap G proteins

1 reference

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Human papillomavirus type 16 E6 and E7 gene variations in Indian cervical cancer

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Human papillomavirus immortalization and transformation functions

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Cell detachment triggers p38 mitogen-activated protein kinase-dependent overexpression of Fas ligand. A novel mechanism of Anoikis of intestinal epithelial cells

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Specific down-modulation of Notch1 signaling in cervical cancer cells is required for sustained HPV-E6/E7 expression and late steps of malignant transformation

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Morphological and biological heterogeneity of three tumorigenic cell lines derived from a single p53-/- osteoblast-like cell line, MMC2.

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Papillomaviruses and cancer: from basic studies to clinical application

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Activated Notch1 signaling cooperates with papillomavirus oncogenes in transformation and generates resistance to apoptosis on matrix withdrawal through PKB/Akt

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Rap1 signalling: adhering to new models

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Transient deactivation of ERK signalling is sufficient for stable entry into G0 in primary avian fibroblasts

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Integrating the MAP kinase signal into the G1 phase cell cycle machinery

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Uneven distribution of HPV 16 E6 prototype and variant (L83V) oncoprotein in cervical neoplastic lesions

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Immortalization of human cells and their malignant conversion by high risk human papillomavirus genotypes

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The Hallmarks of Cancer

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Retention of cell adhesion and growth capability in human cervical cancer cells deprived of cell anchorage

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Creation of human tumour cells with defined genetic elements

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Multiple functions of human papillomavirus type 16 E6 contribute to the immortalization of mammary epithelial cells

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5 July 2018

Pleiotropic effects of human papillomavirus type 16 E6 oncogene expression in human epithelial cell lines

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The human papillomavirus type 16 E6 gene alone is sufficient to induce carcinomas in transgenic animals

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Human ligands of the Notch receptor

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The E6 oncoproteins of high-risk papillomaviruses bind to a novel putative GAP protein, E6TP1, and target it for degradation

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Extracellular signal-regulated activation of Rap1 fails to interfere in Ras effector signalling

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Rap1 mediates sustained MAP kinase activation induced by nerve growth factor

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Human papillomavirus 16 E6 variants are more prevalent in invasive cervical carcinoma than the prototype.

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The ins and outs of notch signaling

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Coassociation of Rap1A and Ha-Ras with Raf-1 N-terminal region interferes with ras-dependent activation of Raf-1

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PI3K: downstream AKTion blocks apoptosis

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Analysis of HLA-DR expression on keratinocytes in cervical neoplasia

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RapV12 antagonizes Ras-dependent activation of ERK1 and ERK2 by LPA and EGF in Rat-1 fibroblasts

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Reversible repression of papillomavirus oncogene expression in cervical carcinoma cells: consequences for the phenotype and E6-p53 and E7-pRB interactions

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Alterations in Notch signaling in neoplastic lesions of the human cervix

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ras oncogenes in human cancer: a review

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Properties of a non-tumorigenic human cervical keratinocyte cell line

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5 July 2018

Characterization of normal human exocervical epithelial cells immortalized in vitro by papillomavirus types 16 and 18 DNA.

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28 October 2018

In vitro and in vivo inhibition of human papillomavirus type 16 E6 and E7 genes

1 reference

https://pubmed.ncbi.nlm.nih.gov/15140991

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

The extracellular signal-regulated kinase pathway is required for activation-induced cell death of T cells

1 reference

https://pubmed.ncbi.nlm.nih.gov/15140991

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Interactions between the PI3K and Raf signaling pathways can result in the transformation of hematopoietic cells

1 reference

https://pubmed.ncbi.nlm.nih.gov/15140991

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Myeloproliferative stem cell disorders by deregulated Rap1 activation in SPA-1-deficient mice

1 reference

https://pubmed.ncbi.nlm.nih.gov/15140991

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

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10.1128/JVI.78.11.5934-5945.2004

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28 December 2019

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28 December 2019

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28 December 2019

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