Control of activating transcription factor 4 (ATF4) persistence by multisite phosphorylation impacts cell cycle progression and neurogenesis. (Q34236497)

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English	Control of activating transcription factor 4 (ATF4) persistence by multisite phosphorylation impacts cell cycle progression and neurogenesis.	scientific article

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10 February 2020

Control of activating transcription factor 4 (ATF4) persistence by multisite phosphorylation impacts cell cycle progression and neurogenesis (English)

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10 February 2020

phosphorylation

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10 February 2020

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Li-Huei Tsai

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10 February 2020

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Christopher L Frank

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10 February 2020

Xuecai Ge

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10 February 2020

Ying Zhou

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19 August 2010

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10 February 2020

Journal of Biological Chemistry

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10 February 2020

285

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43

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10 February 2020

33324-33337

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10 February 2020

Alternative functions of core cell cycle regulators in neuronal migration, neuronal maturation, and synaptic plasticity

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The cell biology of neurogenesis

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ATF4, the osteoblast accumulation of which is determined post-translationally, can induce osteoblast-specific gene expression in non-osteoblastic cells

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The GABA(B) receptor subunits R1 and R2 interact differentially with the activation transcription factor ATF4 in mouse brain during the postnatal development

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An inhibition of cyclin-dependent kinases that lengthens, but does not arrest, neuroepithelial cell cycle induces premature neurogenesis

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Inducible enhancement of memory storage and synaptic plasticity in transgenic mice expressing an inhibitor of ATF4 (CREB-2) and C/EBP proteins

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10.1074/JBC.M110.140699

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