Glycoform-selective prion formation in sporadic and familial forms of prion disease (Q34634279)

3 August 2017

title

Glycoform-selective prion formation in sporadic and familial forms of prion disease (English)

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3 August 2017

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11

Pierluigi Gambetti

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3 August 2017

Xiangzhu Xiao

1

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3 August 2017

Jue Yuan

2

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3 August 2017

Stéphane Haïk

3

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3 August 2017

Ignazio Cali

4

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3 August 2017

Yian Zhan

5

1 reference

3 August 2017

Mohammed Moudjou

6

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3 August 2017

Baiya Li

7

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3 August 2017

Jean-Louis Laplanche

8

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3 August 2017

Hubert Laude

9

1 reference

3 August 2017

Jan Langeveld

10

1 reference

3 August 2017

Tetsuyuki Kitamoto

12

1 reference

3 August 2017

Qingzhong Kong

13

1 reference

3 August 2017

Jean-Philippe Brandel

14

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3 August 2017

Brian A Cobb

15

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3 August 2017

Robert B Petersen

16

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3 August 2017

Wen-Quan Zou

17

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3 August 2017

language of work or name

English

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publication date

19 March 2013

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3 August 2017

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3 August 2017

volume

8

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3 August 2017

issue

3

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3 August 2017

page(s)

e58786

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Creative Commons Attribution 4.0 International

3 August 2017

copyright license

start time

19 March 2013

1 reference

April 2022 Public Data File from Crossref

copyright status

copyrighted

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cites work

A naturally occurring C-terminal fragment of the prion protein (PrP) delays disease and acts as a dominant-negative inhibitor of PrPSc formation.

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Characterization of spontaneously generated prion-like conformers in cultured cells

7 July 2018

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Insoluble cellular prion protein and its association with prion and Alzheimer diseases

7 July 2018

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7 July 2018

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Prion propagation in cells expressing PrP glycosylation mutants

7 July 2018

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Variably protease-sensitive prionopathy: a new sporadic disease of the prion protein.

7 July 2018

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PrP conformational transitions alter species preference of a PrP-specific antibody.

7 July 2018

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Endogenous proteolytic cleavage of disease-associated prion protein to produce C2 fragments is strongly cell- and tissue-dependent

7 July 2018

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7 July 2018

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Trans-dominant inhibition of prion propagation in vitro is not mediated by an accessory cofactor

7 July 2018

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Species and strain glycosylation patterns of PrPSc

7 July 2018

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A novel human disease with abnormal prion protein sensitive to protease

7 July 2018

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Host PrP glycosylation: a major factor determining the outcome of prion infection

7 July 2018

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Accessibility of a critical prion protein region involved in strain recognition and its implications for the early detection of prions

7 July 2018

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A novel phenotype of sporadic Creutzfeldt-Jakob disease.

7 July 2018

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Screening of 145 anti-PrP monoclonal antibodies for their capacity to inhibit PrPSc replication in infected cells.

7 July 2018

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Loss of glycosylation associated with the T183A mutation in human prion disease

7 July 2018

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Glycosylation deficiency at either one of the two glycan attachment sites of cellular prion protein preserves susceptibility to bovine spongiform encephalopathy and scrapie infections.

7 July 2018

1 reference

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Glycan-controlled epitopes of prion protein include a major determinant of susceptibility to sheep scrapie

7 July 2018

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Antibody to DNA detects scrapie but not normal prion protein

7 July 2018

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Dominant-negative inhibition of prion formation diminished by deletion mutagenesis of the prion protein

7 July 2018

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7 July 2018

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Evidence for protein X binding to a discontinuous epitope on the cellular prion protein during scrapie prion propagation

7 July 2018

1 reference

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Heterologous PrP molecules interfere with accumulation of protease-resistant PrP in scrapie-infected murine neuroblastoma cells

7 July 2018

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Mouse polyclonal and monoclonal antibody to scrapie-associated fibril proteins

7 July 2018

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Amyloid protein of Gerstmann-Sträussler-Scheinker disease (Indiana kindred) is an 11 kd fragment of prion protein with an N-terminal glycine at codon 58

7 July 2018

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3602448

V180I mutation of the prion protein gene associated with atypical PrPSc glycosylation.

7 July 2018

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Novel prion protein conformation and glycotype in Creutzfeldt-Jakob disease

21 January 2018

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Truncated forms of the human prion protein in normal brain and in prion diseases.

21 January 2018

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Insoluble aggregates and protease-resistant conformers of prion protein in uninfected human brains.

21 January 2018

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https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0058786

Human prion protein (PrP) 219K is converted to PrPSc but shows heterozygous inhibition in variant Creutzfeldt-Jakob disease infection

21 January 2018

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Dominant-negative effects in prion diseases: insights from molecular dynamics simulations on mouse prion protein chimeras

21 January 2018

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The stoichiometry of host PrPC glycoforms modulates the efficiency of PrPSc formation in vitro.

21 January 2018

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Experimental transmission of Creutzfeldt-Jakob disease and related diseases to rodents

21 January 2018

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The Thr183Ala Mutation, Not the Loss of the First Glycosylation Site, Alters the Physical Properties of the Prion Protein.

21 January 2018

1 reference

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Sequential generation of two structurally distinct ovine prion protein soluble oligomers displaying different biochemical reactivities

26 October 2018

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/23527023

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Effect of the D178N Mutation and the Codon 129 Polymorphism on the Metabolism of the Prion Protein

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/23527023

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Identifiers

DOI

10.1371/JOURNAL.PONE.0058786

1 reference

3 August 2017

0 references

1 reference

3 August 2017

1 reference