Functional complementation between FADD and RIP1 in embryos and lymphocytes (Q34768810)

4 August 2017

Functional complementation between FADD and RIP1 in embryos and lymphocytes (English)

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4 August 2017

Haibing Zhang

1

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4 August 2017

Xiaohui Zhou

2

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4 August 2017

Thomas McQuade

3

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4 August 2017

Jinghe Li

4

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4 August 2017

Francis Ka-Ming Chan

5

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4 August 2017

Jianke Zhang

6

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4 August 2017

language of work or name

English

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publication date

2 March 2011

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4 August 2017

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4 August 2017

471

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4 August 2017

7338

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4 August 2017

373-376

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https://scigraph.springernature.com/pub.10.1038/nature09878

4 August 2017

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cites work

Fas-associated death domain (FADD) is a negative regulator of T-cell receptor-mediated necroptosis

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3072026

Phosphorylation-driven assembly of the RIP1-RIP3 complex regulates programmed necrosis and virus-induced inflammation

7 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3072026

Receptor interacting protein kinase-3 determines cellular necrotic response to TNF-alpha

7 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3072026

RIP3, an energy metabolism regulator that switches TNF-induced cell death from apoptosis to necrosis

7 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3072026

Antigen-mediated T cell expansion regulated by parallel pathways of death

7 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3072026

The Fas-associated death domain protein is required in apoptosis and TLR-induced proliferative responses in B cells

7 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3072026

Chemical inhibitor of nonapoptotic cell death with therapeutic potential for ischemic brain injury

7 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3072026

Essential roles of receptor-interacting protein and TRAF2 in oxidative stress-induced cell death

7 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3072026

The death domain kinase RIP protects thymocytes from tumor necrosis factor receptor type 2-induced cell death

7 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3072026

Fas triggers an alternative, caspase-8-independent cell death pathway using the kinase RIP as effector molecule

7 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3072026

Targeted disruption of the mouse Caspase 8 gene ablates cell death induction by the TNF receptors, Fas/Apo1, and DR3 and is lethal prenatally.

7 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3072026

The death domain kinase RIP mediates the TNF-induced NF-kappaB signal

7 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3072026

Fas-mediated apoptosis and activation-induced T-cell proliferation are defective in mice lacking FADD/Mort1

7 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3072026

FADD: essential for embryo development and signaling from some, but not all, inducers of apoptosis

7 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3072026

Apoptosis by death factor

7 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3072026

FLICE, a novel FADD-homologous ICE/CED-3-like protease, is recruited to the CD95 (Fas/APO-1) death--inducing signaling complex

7 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3072026

Involvement of MACH, a novel MORT1/FADD-interacting protease, in Fas/APO-1- and TNF receptor-induced cell death

7 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3072026

A mouse Fas-associated protein with homology to the human Mort1/FADD protein is essential for Fas-induced apoptosis

7 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3072026

FADD, a novel death domain-containing protein, interacts with the death domain of Fas and initiates apoptosis

7 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3072026

A novel protein that interacts with the death domain of Fas/APO1 contains a sequence motif related to the death domain

7 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3072026

Conditional Fas-associated death domain protein (FADD): GFP knockout mice reveal FADD is dispensable in thymic development but essential in peripheral T cell homeostasis

7 July 2018

1 reference

Crossref

https://api.crossref.org/works/10.1038%2FNATURE09878

A role for tumor necrosis factor receptor-2 and receptor-interacting protein in programmed necrosis and antiviral responses

21 January 2018

1 reference

Crossref

https://api.crossref.org/works/10.1038%2FNATURE09878

FADD deficiency impairs early hematopoiesis in the bone marrow

21 January 2018

1 reference

Crossref

https://api.crossref.org/works/10.1038%2FNATURE09878

21 January 2018

Identifiers

DOI

10.1038/NATURE09878

1 reference

Dimensions Publication ID

4 August 2017

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4 August 2017

1 reference