Receptor interacting protein kinase-3 determines cellular necrotic response to TNF-alpha (Q24338113)

2

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Lin Miao

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Tao Wang

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Fenghe Du

5

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Liping Zhao

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Xiaodong Wang

Necrosis, a well-orchestrated form of cell demise: signalling cascades, important mediators and concomitant immune response

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language of work or name

English

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publication date

12 June 2009

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published in

Cell

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volume

137

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issue

6

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page(s)

1100-11

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cites work

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2009.05.021

7 January 2021

Autocrine TNFalpha signaling renders human cancer cells susceptible to Smac-mimetic-induced apoptosis

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2009.05.021

7 January 2021

IAP antagonists induce autoubiquitination of c-IAPs, NF-kappaB activation, and TNFalpha-dependent apoptosis

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2009.05.021

7 January 2021

IAP antagonists target cIAP1 to induce TNFalpha-dependent apoptosis

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2009.05.021

7 January 2021

TNF-alpha induces two distinct caspase-8 activation pathways

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2009.05.021

7 January 2021

Identification of a molecular signaling network that regulates a cellular necrotic cell death pathway

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2009.05.021

7 January 2021

TNF-induced activation of the Nox1 NADPH oxidase and its role in the induction of necrotic cell death

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2009.05.021

7 January 2021

Smac, a mitochondrial protein that promotes cytochrome c-dependent caspase activation by eliminating IAP inhibition

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2009.05.021

7 January 2021

Identification of DIABLO, a mammalian protein that promotes apoptosis by binding to and antagonizing IAP proteins

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2009.05.021

7 January 2021

Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2009.05.021

7 January 2021

Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2009.05.021

7 January 2021

Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2009.05.021

7 January 2021

Identification of RIP3, a RIP-like kinase that activates apoptosis and NFkappaB

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2009.05.021

7 January 2021

TNF-dependent recruitment of the protein kinase RIP to the TNF receptor-1 signaling complex

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2009.05.021

7 January 2021

The death domain kinase RIP mediates the TNF-induced NF-kappaB signal

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2009.05.021

7 January 2021

Fas triggers an alternative, caspase-8-independent cell death pathway using the kinase RIP as effector molecule

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2009.05.021

7 January 2021

Apoptosis: A Basic Biological Phenomenon with Wideranging Implications in Tissue Kinetics

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2009.05.021

7 January 2021

Cyclophilin D-dependent mitochondrial permeability transition regulates some necrotic but not apoptotic cell death

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2009.05.021

7 January 2021

Chemical inhibitor of nonapoptotic cell death with therapeutic potential for ischemic brain injury

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2009.05.021

7 January 2021

Identification of RIP1 kinase as a specific cellular target of necrostatins

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2009.05.021

7 January 2021

The CD95(APO-1/Fas) DISC and beyond

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2009.05.021

7 January 2021

RIP3, a novel apoptosis-inducing kinase

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2009.05.021

7 January 2021

Identification of a novel homotypic interaction motif required for the phosphorylation of receptor-interacting protein (RIP) by RIP3.

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2009.05.021

7 January 2021

A role for tumor necrosis factor receptor-2 and receptor-interacting protein in programmed necrosis and antiviral responses

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2009.05.021

7 January 2021

Tumor necrosis factor-induced nonapoptotic cell death requires receptor-interacting protein-mediated cellular reactive oxygen species accumulation

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2009.05.021

7 January 2021

Cell death in pancreatitis: caspases protect from necrotizing pancreatitis

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2009.05.021

7 January 2021

Inhibition of caspases increases the sensitivity of L929 cells to necrosis mediated by tumor necrosis factor.

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2009.05.021

7 January 2021

Dual signaling of the Fas receptor: initiation of both apoptotic and necrotic cell death pathways

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2009.05.021

7 January 2021

Cleavage of the death domain kinase RIP by caspase-8 prompts TNF-induced apoptosis

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2009.05.021

7 January 2021

Cell death in health and disease

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2009.05.021

7 January 2021

A small molecule Smac mimic potentiates TRAIL- and TNFalpha-mediated cell death

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2009.05.021

7 January 2021

Caspases: enemies within

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2009.05.021

7 January 2021

Kinase RIP3 is dispensable for normal NF-kappa Bs, signaling by the B-cell and T-cell receptors, tumor necrosis factor receptor 1, and Toll-like receptors 2 and 4.

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2009.05.021

7 January 2021

Inhibition of ADP/ATP exchange in receptor-interacting protein-mediated necrosis

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2009.05.021

7 January 2021