Significance and mechanism of Alzheimer neurofibrillary degeneration and therapeutic targets to inhibit this lesion (Q34814821)

4 August 2017

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title

Significance and mechanism of Alzheimer neurofibrillary degeneration and therapeutic targets to inhibit this lesion (English)

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4 August 2017

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Inge Grundke-Iqbal

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4 August 2017

4

Cheng-Xin Gong

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4 August 2017

Khalid Iqbal

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Alejandra del C Alonso

2

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Ezzat El-Akkad

3

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Niloufar Haque

5

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Sabiha Khatoon

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Jin-Jing Pei

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Ichiro Tsujio

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Jian-Zhi Wang

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1 August 2002

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Journal of Molecular Neuroscience

4 August 2017

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4 August 2017

19

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95-99

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1-2

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https://scigraph.springernature.com/pub.10.1007/s12031-002-0017-3

4 August 2017

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Can, a putative oncogene associated with myeloid leukemogenesis, may be activated by fusion of its 3' half to different genes: characterization of the set gene

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Alzheimer's disease hyperphosphorylated tau sequesters normal tau into tangles of filaments and disassembles microtubules.

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Mechanism of neurofibrillary degeneration and pharmacologic therapeutic approach.

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Role of abnormally phosphorylated tau in the breakdown of microtubules in Alzheimer disease

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Hyperphosphorylation induces self-assembly of tau into tangles of paired helical filaments/straight filaments

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Accumulation of abnormally phosphorylated tau precedes the formation of neurofibrillary tangles in Alzheimer's disease

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Tau is a candidate gene for chromosome 17 frontotemporal dementia

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Alzheimer paired helical filaments. Restoration of the biological activity by dephosphorylation.

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12 December 2020

10.1007/S12031-002-0017-3

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DOI

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Dimensions Publication ID

4 August 2017

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