Mitochondrial c-Jun N-terminal kinase (JNK) signaling initiates physiological changes resulting in amplification of reactive oxygen species generation. (Q34963377)

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English	Mitochondrial c-Jun N-terminal kinase (JNK) signaling initiates physiological changes resulting in amplification of reactive oxygen species generation.	scientific article

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27 January 2020

Mitochondrial c-Jun N-terminal kinase (JNK) signaling initiates physiological changes resulting in amplification of reactive oxygen species generation (English)

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27 January 2020

reactive oxygen species

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Jeremy W. Chambers

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Philip V LoGrasso

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16 March 2011

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27 January 2020

Journal of Biological Chemistry

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16052-16062

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27 January 2020

Regulation of mitochondrial glutathione redox status and protein glutathionylation by respiratory substrates

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8 July 2018

Mkp1 is a c-Jun target gene that antagonizes JNK-dependent apoptosis in sympathetic neurons

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8 July 2018

Mitochondrial reactive oxygen species regulate cellular signaling and dictate biological outcomes

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8 July 2018

Detection and manipulation of mitochondrial reactive oxygen species in mammalian cells

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Synthesis, Biological Evaluation, X-ray Structure, and Pharmacokinetics of Aminopyrimidine c-jun-N-terminal Kinase (JNK) Inhibitors

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8 July 2018

Prevention of premature senescence requires JNK regulation of Bcl-2 and reactive oxygen species

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8 July 2018

Mammalian mitochondrial complex I: biogenesis, regulation, and reactive oxygen species generation.

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Signal integration by JNK and p38 MAPK pathways in cancer development

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8 July 2018

Cerebral ischemia provokes a profound exchange of activated JNK isoforms in brain mitochondria

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8 July 2018

How mitochondria produce reactive oxygen species

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8 July 2018

Mitochondrial complex III-generated oxidants activate ASK1 and JNK to induce alveolar epithelial cell death following exposure to particulate matter air pollution

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8 July 2018

JNK signaling in apoptosis

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8 July 2018

Role of p38 MAPK and JNK in enhanced cervical cancer cell killing by the combination of arsenic trioxide and ionizing radiation

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8 July 2018

Positioning mitochondrial plasticity within cellular signaling cascades.

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8 July 2018

The role of p38 MAPK and JNK in Arsenic trioxide-induced mitochondrial cell death in human cervical cancer cells

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8 July 2018

Role of JNK translocation to mitochondria leading to inhibition of mitochondria bioenergetics in acetaminophen-induced liver injury

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Effects of 4-hydroxynonenal on mitochondrial 3-hydroxy-3-methylglutaryl (HMG-CoA) synthase

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Simultaneous detection of apoptosis and mitochondrial superoxide production in live cells by flow cytometry and confocal microscopy

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8 July 2018

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Isolation and subfractionation of mitochondria from animal cells and tissue culture lines.

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8 July 2018

Mitochondria are a major source of paraquat-induced reactive oxygen species production in the brain

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8 July 2018

Superoxide flux in endothelial cells via the chloride channel-3 mediates intracellular signaling

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8 July 2018

Generation of superoxide by the mitochondrial Complex I.

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8 July 2018

JNK signaling pathway is a key modulator in cell death mediated by reactive oxygen and nitrogen species

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8 July 2018

Mitochondrial modulation: reversible phosphorylation takes center stage?

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8 July 2018

Glutathionylation of mitochondrial proteins

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8 July 2018

Reactive oxygen species promote TNFalpha-induced death and sustained JNK activation by inhibiting MAP kinase phosphatases

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Kinase signaling cascades in the mitochondrion: a matter of life or death.

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8 July 2018

JNK potentiates TNF-stimulated necrosis by increasing the production of cytotoxic reactive oxygen species

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8 July 2018

Sab (SH3BP5), a novel mitochondria-localized JNK-interacting protein.

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8 July 2018

Mitochondrial signals initiate the activation of c-Jun N-terminal kinase (JNK) by hypoxia-reoxygenation

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The JNK cascade as a biochemical switch in mammalian cells: ultrasensitive and all-or-none responses

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8 July 2018

A new c-Jun N-terminal kinase (JNK)-interacting protein, Sab (SH3BP5), associates with mitochondria

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The Bax subfamily of Bcl2-related proteins is essential for apoptotic signal transduction by c-Jun NH(2)-terminal kinase

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Role for mitochondrial oxidants as regulators of cellular metabolism

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Purification of a reconstitutively active iron-sulfur protein (oxidation factor) from succinate . cytochrome c reductase complex of bovine heart mitochondria

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ASK1 is required for sustained activations of JNK/p38 MAP kinases and apoptosis

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Mitochondrial targeting of JNK/SAPK in the phorbol ester response of myeloid leukemia cells

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Amplification loop cascade for increasing caspase activity induced by docetaxel

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Oxidative damage to mitochondria is a preliminary step to caspase-3 activation in fluoride-induced apoptosis in HL-60 cells

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c-Jun N-terminal kinase regulates mitochondrial bioenergetics by modulating pyruvate dehydrogenase activity in primary cortical neurons

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Characteristics of the inhibition of NADPH oxidase activation in neutrophils by apocynin, a methoxy-substituted catechol

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Chemical genetic analysis of the time course of signal transduction by JNK

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The JNK signal transduction pathway

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12 December 2020

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Ionizing radiation utilizes c-Jun N-terminal kinase for amplification of mitochondrial apoptotic cell death in human cervical cancer cells

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Translocation of SAPK/JNK to mitochondria and interaction with Bcl-x(L) in response to DNA damage

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Direct activation of mitochondrial apoptosis machinery by c-Jun N-terminal kinase in adult cardiac myocytes

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Identifiers

10.1074/JBC.M111.223602

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27 January 2020

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27 January 2020

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27 January 2020

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