Exenatide protects against hypoxia/reoxygenation-induced apoptosis by improving mitochondrial function in H9c2 cells. (Q35105534)

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9 March 2020

title

Exenatide protects against hypoxia/reoxygenation-induced apoptosis by improving mitochondrial function in H9c2 cells (English)

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9 March 2020

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6

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9 March 2020

author name string

Guanglei Chang

1

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9 March 2020

Dongying Zhang

2

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9 March 2020

Jian Liu

3

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9 March 2020

Peng Zhang

4

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9 March 2020

Lin Ye

5

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9 March 2020

Qin Duan

7

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9 March 2020

Aihua Zheng

8

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Shu Qin

9

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publication date

28 February 2014

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Experimental Biology and Medicine

9 March 2020

published in

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9 March 2020

volume

239

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9 March 2020

issue

4

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9 March 2020

page(s)

414-422

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Ventricular remodeling after myocardial infarction. Experimental observations and clinical implications

9 March 2020

cites work

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Exenatide reduces infarct size and improves cardiac function in a porcine model of ischemia and reperfusion injury

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Antiapoptotic effects of GLP-1 in murine HL-1 cardiomyocytes

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Globular adiponectin, acting via AdipoR1/APPL1, protects H9c2 cells from hypoxia/reoxygenation-induced apoptosis

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Differentiation of human bone marrow mesenchymal stem cells grown in terpolyesters of 3-hydroxyalkanoates scaffolds into nerve cells

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High glucose-induced Ca2+ overload and oxidative stress contribute to apoptosis of cardiac cells through mitochondrial dependent and independent pathways

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

Identifiers

DOI

10.1177/1535370214522177

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:24586099%20AND%20SRC:MED&resulttype=core&format=json

9 March 2020

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