Differential effects of buffer pH on Ca(2+)-induced ROS emission with inhibited mitochondrial complexes I and III. (Q35161240)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:25805998%20AND%20SRC:MED&resulttype=core&format=json

3 March 2020

title

Differential effects of buffer pH on Ca(2+)-induced ROS emission with inhibited mitochondrial complexes I and III (English)

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3 March 2020

3

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3 March 2020

author name string

Daniel P Lindsay

1

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3 March 2020

Amadou K S Camara

2

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3 March 2020

Ryan Lubbe

4

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3 March 2020

Mohammed Aldakkak

5

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publication date

10 March 2015

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3 March 2020

published in

Frontiers in Physiology

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3 March 2020

volume

6

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3 March 2020

page(s)

58

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Ischaemic accumulation of succinate controls reperfusion injury through mitochondrial ROS.

3 March 2020

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Mitochondrial ROS-induced ROS release: an update and review

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Mitochondrial calcium and oxidative stress as mediators of ischemic brain injury.

13 July 2018

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New concepts in reactive oxygen species and cardiovascular reperfusion physiology

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13 July 2018

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Possible occurrence and role of an essential histidyl residue in succinate dehydrogenase.

13 July 2018

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Calcium-dependent opening of a non-specific pore in the mitochondrial inner membrane is inhibited at pH values below 7. Implications for the protective effect of low pH against chemical and hypoxic cell damage

13 July 2018

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27 September 2018

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27 September 2018

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27 September 2018

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Multiple Q-cycle bypass reactions at the Qo site of the cytochrome bc1 complex

27 September 2018

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Generation of superoxide in cardiomyocytes during ischemia before reperfusion.

27 September 2018

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The Ca2+-induced membrane transition in mitochondria

27 September 2018

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Crossref

https://api.crossref.org/works/10.3389%2FFPHYS.2015.00058

Ubisemiquinone is the electron donor for superoxide formation by complex III of heart mitochondria

21 January 2018

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12 December 2020

The inhibition of succinate dehydrogenase by oxalacetate

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12 December 2020

ROS scavenging before 27 degrees C ischemia protects hearts and reduces mitochondrial ROS, Ca2+ overload, and changes in redox state

1 reference

12 December 2020

Ischemic injury to mitochondrial electron transport in the aging heart: damage to the iron-sulfur protein subunit of electron transport complex III

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12 December 2020

Changes in intracellular Na+ and pH in rat heart during ischemia: role of Na+/H+ exchanger

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12 December 2020

Significant levels of oxidants are generated by isolated cardiomyocytes during ischemia prior to reperfusion

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12 December 2020

Interaction of the membrane-bound succinate dehydrogenase with substrate and competitive inhibitors

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12 December 2020

Cytosolic calcium in the ischemic rabbit heart: assessment by pH- and temperature-adjusted rhod-2 spectrofluorometry

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12 December 2020

Reduced reactive O2 species formation and preserved mitochondrial NADH and [Ca2+] levels during short-term 17 degrees C ischemia in intact hearts

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12 December 2020

Mitochondrial complexes I, II, III, IV, and V in myocardial ischemia and autolysis

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12 December 2020

Modulation of the mitochondrial permeability transition pore. Effect of protons and divalent cations

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12 December 2020

Identifiers

DOI

10.3389/FPHYS.2015.00058

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:25805998%20AND%20SRC:MED&resulttype=core&format=json

3 March 2020

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:25805998%20AND%20SRC:MED&resulttype=core&format=json

3 March 2020

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:25805998%20AND%20SRC:MED&resulttype=core&format=json