The requirement for evasion of programmed cell death in neuroblastomas with MYCN amplification (Q35184984)

7 August 2017

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title

The requirement for evasion of programmed cell death in neuroblastomas with MYCN amplification (English)

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7 August 2017

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Michael D Hogarty

1

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7 August 2017

language of work or name

English

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publication date

1 July 2003

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7 August 2017

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7 August 2017

volume

197

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7 August 2017

issue

1-2

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7 August 2017

page(s)

173-179

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Infant cancer in the U.S.: histology-specific incidence and trends, 1973 to 1992.

7 August 2017

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Requirement for the CD95 receptor-ligand pathway in c-Myc-induced apoptosis

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Apoptosis in the nervous system

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7 January 2021

Targeted expression of MYCN causes neuroblastoma in transgenic mice

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7 January 2021

Disruption of the ARF-Mdm2-p53 tumor suppressor pathway in Myc-induced lymphomagenesis

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7 January 2021

Caspase 8 is deleted or silenced preferentially in childhood neuroblastomas with amplification of MYCN

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7 January 2021

IGF-I receptor activation and BCL-2 overexpression prevent early apoptotic events in human neuroblastoma

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7 January 2021

Trk receptor tyrosine kinases: a bridge between cancer and neural development

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7 January 2021

Expression of the Gene for Multidrug-Resistance–Associated Protein and Outcome in Patients with Neuroblastoma

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7 January 2021

Bcl-2 mediates chemoresistance in matched pairs of primary E(mu)-myc lymphomas in vivo

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7 January 2021

The p16 (CDKN2A) gene is involved in the growth of neuroblastoma cells and its expression is associated with prognosis of neuroblastoma patients

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7 January 2021

BIN1 inhibits colony formation and induces apoptosis in neuroblastoma cell lines with MYCN amplification

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7 January 2021

Caspase-9 and Apaf-1 are expressed and functionally active in human neuroblastoma tumor cell lines with 1p36 LOH and amplified MYCN.

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7 January 2021

High expression of Survivin, mapped to 17q25, is significantly associated with poor prognostic factors and promotes cell survival in human neuroblastoma

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7 January 2021

10.1016/S0304-3835(03)00103-4

Identifiers

DOI

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7 August 2017

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