Mice overexpressing BAFF develop a commensal flora-dependent, IgA-associated nephropathy (Q35369982)

8 August 2017

title

Mice overexpressing BAFF develop a commensal flora-dependent, IgA-associated nephropathy (English)

1 reference

8 August 2017

1 reference

Jeffrey L Browning

18

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Jennifer L Gommerman

object named as

Jennifer L Gommerman

17

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author name string

Douglas D McCarthy

1

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8 August 2017

Julie Kujawa

2

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8 August 2017

Cheryl Wilson

3

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8 August 2017

Adrian Papandile

4

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8 August 2017

Urjana Poreci

5

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8 August 2017

Elisa A Porfilio

6

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8 August 2017

Lesley Ward

7

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8 August 2017

Melissa A E Lawson

8

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8 August 2017

Andrew J Macpherson

9

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8 August 2017

Kathy D McCoy

10

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8 August 2017

York Pei

11

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8 August 2017

Lea Novak

12

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8 August 2017

Jeannette Y Lee

13

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8 August 2017

Bruce A Julian

14

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8 August 2017

Jan Novak

15

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8 August 2017

Ann Ranger

16

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8 August 2017

language of work or name

English

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publication date

1 September 2011

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Journal of Clinical Investigation

8 August 2017

published in

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8 August 2017

volume

121

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8 August 2017

issue

10

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8 August 2017

page(s)

3991-4002

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Update on mucosal immunoglobulin A in gastrointestinal disease

8 August 2017

cites work

1 reference

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Aberrantly glycosylated IgA1 in IgA nephropathy patients is recognized by IgG antibodies with restricted heterogeneity

27 July 2018

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Pathogenetic significance of aberrant glycosylation of IgA1 in IgA nephropathy

27 July 2018

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27 July 2018

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IL5RA and TNFRSF6B gene variants are associated with sporadic IgA nephropathy

27 July 2018

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Potential immunopathogenic role of the mucosa-bone marrow axis in IgA nephropathy: insights from animal models

27 July 2018

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TACI, unlike BAFF-R, is solely activated by oligomeric BAFF and APRIL to support survival of activated B cells and plasmablasts

27 July 2018

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BAFF and MyD88 signals promote a lupuslike disease independent of T cells

27 July 2018

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27 July 2018

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27 July 2018

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27 July 2018

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27 July 2018

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27 July 2018

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Dysregulated LIGHT expression on T cells mediates intestinal inflammation and contributes to IgA nephropathy

27 July 2018

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27 July 2018

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Mice transgenic for BAFF develop lymphocytic disorders along with autoimmune manifestations

27 July 2018

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27 July 2018

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27 July 2018

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Structural features of IgA molecules which contribute to IgA nephropathy.

27 July 2018

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27 July 2018

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IgA glycosylation and IgA immune complexes in the pathogenesis of IgA nephropathy

27 July 2018

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Alternative and classical NF-kappa B signaling retain autoreactive B cells in the splenic marginal zone and result in lupus-like disease

26 September 2018

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Development of nephritis but not sialadenitis in autoimmune-prone BAFF transgenic mice lacking marginal zone B cells.

26 September 2018

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26 September 2018

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26 September 2018

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Inhibition of B cell death causes the development of an IgA nephropathy in (New Zealand white x C57BL/6)F(1)-bcl-2 transgenic mice.

26 September 2018

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26 September 2018

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CTLA4Ig alters the course of autoimmune disease development in Lyn-/- mice

26 September 2018

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Regional variations in the distributions of small intestinal intraepithelial lymphocytes in germ-free and specific pathogen-free mice

24 October 2018

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12 December 2020

Signaling via LTbetaR on the lamina propria stromal cells of the gut is required for IgA production

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12 December 2020

TNF Deficiency Fails to Protect BAFF Transgenic Mice against Autoimmunity and Reveals a Predisposition to B Cell Lymphoma

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CR1/CR2 deficiency alters IgG3 autoantibody production and IgA glomerular deposition in the MRL/lpr model of SLE

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C4 in glomerular lesions of NZB/NZW mice

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Targeted deletion of the IgA constant region in mice leads to IgA deficiency with alterations in expression of other Ig isotypes

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12 December 2020

BAFF induces a hyper-IgA syndrome in the intestinal lamina propria concomitant with IgA deposition in the kidney independent of LIGHT

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Differential glycosylation of polymeric and monomeric IgA: a possible role in glomerular inflammation in IgA nephropathy

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12 December 2020

[Therapeutic effects and prognostic factors in tonsillectomy patients with IgA nephropathy]

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12 December 2020

TACI regulates IgA production by APRIL in collaboration with HSPG

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12 December 2020

Intestinal Bacteria Trigger T Cell-Independent Immunoglobulin A2 Class Switching by Inducing Epithelial-Cell Secretion of the Cytokine APRIL

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12 December 2020

Increase in B-cell-activation factor (BAFF) and IFN-gamma productions by tonsillar mononuclear cells stimulated with deoxycytidyl-deoxyguanosine oligodeoxynucleotides (CpG-ODN) in patients with IgA nephropathy

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Identifiers

DOI

10.1172/JCI45563

1 reference

8 August 2017

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8 August 2017

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