Autophagic degradation of farnesylated prelamin A as a therapeutic approach to lamin-linked progeria. (Q35771419)

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21 January 2020

title

Autophagic degradation of farnesylated prelamin A as a therapeutic approach to lamin-linked progeria (English)

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21 January 2020

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21 January 2020

Cristina Capanni

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Marta Columbaro

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Giuseppe Novelli

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Milena Fini

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Sandra Marmiroli

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Stefano Squarzoni

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Giovanna Lattanzi

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21 January 2020

author name string

M Ortolani

4

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M R D'Apice

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E Scarano

9

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N M Maraldi

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S Prencipe

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publication date

19 October 2011

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European Journal of Histochemistry

21 January 2020

published in

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volume

55

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21 January 2020

issue

4

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21 January 2020

page(s)

e36

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Identification of differential protein interactors of lamin A and progerin

21 January 2020

cites work

1 reference

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Mammalian SUN protein interaction networks at the inner nuclear membrane and their role in laminopathy disease processes

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Mutant nuclear lamin A leads to progressive alterations of epigenetic control in premature aging

27 July 2018

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Nuclear envelope proteins and chromatin arrangement: a pathogenic mechanism for laminopathies.

27 July 2018

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Rescue of heterochromatin organization in Hutchinson-Gilford progeria by drug treatment

27 July 2018

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Reversal of the cellular phenotype in the premature aging disease Hutchinson-Gilford progeria syndrome

27 July 2018

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LAP2alpha and BAF transiently localize to telomeres and specific regions on chromatin during nuclear assembly

27 July 2018

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Accumulation of mutant lamin A causes progressive changes in nuclear architecture in Hutchinson-Gilford progeria syndrome

27 July 2018

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Prelamin A-mediated recruitment of SUN1 to the nuclear envelope directs nuclear positioning in human muscle.

27 July 2018

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Lamin A precursor induces barrier-to-autointegration factor nuclear localization.

25 September 2018

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Different prelamin A forms accumulate in human fibroblasts: a study in experimental models and progeria.

25 September 2018

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Eliminating the synthesis of mature lamin A reduces disease phenotypes in mice carrying a Hutchinson-Gilford progeria syndrome allele.

25 September 2018

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Pre-Lamin A processing is linked to heterochromatin organization.

25 September 2018

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Hutchinson-Gilford progeria syndrome: review of the phenotype.

25 September 2018

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Novel lamin A/C gene (LMNA) mutations in atypical progeroid syndromes

25 September 2018

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Recurrent de novo point mutations in lamin A cause Hutchinson–Gilford progeria syndrome

25 September 2018

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Osteoblasts from a mandibuloacral dysplasia patient induce human blood precursors to differentiate into active osteoclasts

25 September 2018

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Autophagy is defective in collagen VI muscular dystrophies and its reactivation rescues myofiber degeneration

25 September 2018

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Site-dependent differences in both prelamin A and adipogenic genes in subcutaneous adipose tissue of patients with type 2 familial partial lipodystrophy

15 November 2018

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/22297442

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Identifiers

DOI

10.4081/EJH.2011.E36

1 reference

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21 January 2020

1 reference

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21 January 2020

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