Local delivery of AAV2-CTLA4IgG decreases sialadenitis and improves gland function in the C57BL/6.NOD-Aec1Aec2 mouse model of Sjögren's syndrome (Q36085054)

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English	Local delivery of AAV2-CTLA4IgG decreases sialadenitis and improves gland function in the C57BL/6.NOD-Aec1Aec2 mouse model of Sjögren's syndrome	scientific article

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scholarly article

1 reference

Europe PubMed Central

PMC publication ID

12 August 2017

Local delivery of AAV2-CTLA4IgG decreases sialadenitis and improves gland function in the C57BL/6.NOD-Aec1Aec2 mouse model of Sjögren's syndrome (English)

1 reference

Europe PubMed Central

PMC publication ID

12 August 2017

author name string

Hongen Yin

1

1 reference

Europe PubMed Central

PMC publication ID

12 August 2017

Cuong Q Nguyen

2

1 reference

Europe PubMed Central

PMC publication ID

12 August 2017

Yuval Samuni

3

1 reference

Europe PubMed Central

PMC publication ID

12 August 2017

Toshimitsu Uede

4

1 reference

Europe PubMed Central

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12 August 2017

Ammon B Peck

5

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Europe PubMed Central

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12 August 2017

John A Chiorini

6

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12 August 2017

publication date

27 February 2012

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Europe PubMed Central

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12 August 2017

Arthritis Research and Therapy

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Europe PubMed Central

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12 August 2017

14

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Europe PubMed Central

PMC publication ID

12 August 2017

1

1 reference

Europe PubMed Central

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12 August 2017

R40

1 reference

Europe PubMed Central

PMC publication ID

12 August 2017

Gene delivery in salivary glands: from the bench to the clinic

1 reference

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Pathogenic effect of interleukin-17A in induction of Sjögren's syndrome-like disease using adenovirus-mediated gene transfer

1 reference

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Pathogenetic mechanisms in the initiation and perpetuation of Sjögren's syndrome

1 reference

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The cytokine milieu in the interplay of pathogenic Th1/Th17 cells and regulatory T cells in autoimmune disease

1 reference

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Interleukin-12 induces salivary gland dysfunction in transgenic mice, providing a new model of Sjögren's syndrome.

1 reference

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CTLA4-Ig interacts with cultured synovial macrophages from rheumatoid arthritis patients and downregulates cytokine production

1 reference

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Differential gene expression in the salivary gland during development and onset of xerostomia in Sjögren's syndrome-like disease of the C57BL/6.NOD-Aec1Aec2 mouse

1 reference

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Salivary gland tissue expression of interleukin-23 and interleukin-17 in Sjögren's syndrome: findings in humans and mice

1 reference

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1 reference

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Reengineered salivary glands are stable endogenous bioreactors for systemic gene therapeutics

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Adeno-associated virus serotype 4 (AAV4) and AAV5 both require sialic acid binding for hemagglutination and efficient transduction but differ in sialic acid linkage specificity

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Glandular and extraglandular expression of costimulatory molecules in patients with Sjögren's syndrome

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Immunologic self-tolerance maintained by CD25(+)CD4(+) regulatory T cells constitutively expressing cytotoxic T lymphocyte-associated antigen 4.

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Interferon-gamma induces persistent depletion of internal Ca2+ stores in a human salivary gland cell line

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Regulation of CTLA-4 expression during T cell activation

1 reference

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The histopathology of Sjögren's syndrome in labial salivary gland biopsies

1 reference

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Enhanced transduction of mouse salivary glands with AAV5-based vectors

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T cell subpopulations in the labial minor salivary gland histopathologic lesion of Sjögren's syndrome

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Influence of CTLA4 haplotypes on susceptibility and some extraglandular manifestations in primary Sjögren's syndrome.

1 reference

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Combined gene therapy with adenovirus vectors containing CTLA4Ig and CD40Ig prolongs survival of composite tissue allografts in rat model

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Autoimmune Sjögren's-like lesions in salivary glands of TGF-beta1-deficient mice are inhibited by adhesion-blocking peptides.

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CD4 cytotoxic and dendritic cells in the immunopathologic lesion of Sjögren's syndrome

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Two NOD Idd-associated intervals contribute synergistically to the development of autoimmune exocrinopathy (Sjögren's syndrome) on a healthy murine background

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Possible function of salivary gland epithelial cells as nonprofessional antigen-presenting cells in the development of Sjögren's syndrome

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Serological diagnosis of primary Sjögren's syndrome by means of human recombinant La (SS-B) as nuclear antigen

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Abatacept for rheumatoid arthritis refractory to tumor necrosis factor alpha inhibition

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Cre/loxP-mediated CTLA4IgG gene transfer induces clinically relevant immunosuppression via on-off gene recombination in vivo

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T regulatory cells are markedly diminished in diseased salivary glands of patients with primary Sjögren's syndrome

1 reference

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12 August 2017

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12 August 2017

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