Cyclosporin A inhibits caspase-independent death of NGF-deprived sympathetic neurons: a potential role for mitochondrial permeability transition (Q36324351)

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English	Cyclosporin A inhibits caspase-independent death of NGF-deprived sympathetic neurons: a potential role for mitochondrial permeability transition	scientific article

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scholarly article

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

Cyclosporin A inhibits caspase-independent death of NGF-deprived sympathetic neurons: a potential role for mitochondrial permeability transition (English)

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

1 reference

ChEBI release 2020-05-28

author name string

Louis K Chang

1

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

Eugene M Johnson

2

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Europe PubMed Central

PMC publication ID

13 August 2017

language of work or name

0 references

publication date

20 May 2002

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Europe PubMed Central

PMC publication ID

13 August 2017

Journal of Cell Biology

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Europe PubMed Central

PMC publication ID

13 August 2017

157

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

5

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

771-781

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

A serine protease, HtrA2, is released from the mitochondria and interacts with XIAP, inducing cell death

1 reference

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30 September 2017

Endonuclease G is an apoptotic DNase when released from mitochondria

1 reference

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30 September 2017

Cytochrome c maintains mitochondrial transmembrane potential and ATP generation after outer mitochondrial membrane permeabilization during the apoptotic process

1 reference

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30 September 2017

Essential role of the mitochondrial apoptosis-inducing factor in programmed cell death

1 reference

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30 September 2017

Two distinct action mechanisms of immunophilin-ligand complexes for the blockade of T-cell activation

1 reference

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30 September 2017

Permeabilization of the mitochondrial inner membrane during apoptosis: impact of the adenine nucleotide translocator

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2173417

30 September 2017

Caspase inhibition extends the commitment to neuronal death beyond cytochrome c release to the point of mitochondrial depolarization

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2173417

30 September 2017

Mechanisms of action of cyclosporine.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2173417

30 September 2017

Inhibition of apoptotic signaling cascades causes loss of trophic factor dependence during neuronal maturation

1 reference

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30 September 2017

Electrophysiological study of a novel large pore formed by Bax and the voltage-dependent anion channel that is permeable to cytochrome c.

1 reference

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30 September 2017

The mitochondrial permeability transition pore and its role in cell death

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2173417

30 September 2017

Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC

1 reference

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30 September 2017

Mitochondrial depolarization accompanies cytochrome c release during apoptosis in PC6 cells

1 reference

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30 September 2017

Molecular characterization of mitochondrial apoptosis-inducing factor

1 reference

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30 September 2017

The mitochondrial permeability transition is required for tumor necrosis factor alpha-mediated apoptosis and cytochrome c release

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2173417

30 September 2017

Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

1 reference

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30 September 2017

Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

1 reference

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30 September 2017

Mitochondrial cytochrome c release in apoptosis occurs upstream of DEVD-specific caspase activation and independently of mitochondrial transmembrane depolarization.

1 reference

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30 September 2017

Bcl-xL regulates the membrane potential and volume homeostasis of mitochondria

1 reference

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30 September 2017

Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

1 reference

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30 September 2017

Bax deletion further orders the cell death pathway in cerebellar granule cells and suggests a caspase-independent pathway to cell death.

1 reference

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30 September 2017

Mitochondrial permeability transition is a central coordinating event of apoptosis

1 reference

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30 September 2017

Inhibition of interleukin 1beta converting enzyme family proteases reduces ischemic and excitotoxic neuronal damage

1 reference

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30 September 2017

Genetic and metabolic status of NGF-deprived sympathetic neurons saved by an inhibitor of ICE family proteases

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2173417

30 September 2017

BAX is required for neuronal death after trophic factor deprivation and during development

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2173417

30 September 2017

FLICE, a novel FADD-homologous ICE/CED-3-like protease, is recruited to the CD95 (Fas/APO-1) death--inducing signaling complex

1 reference

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30 September 2017

The contrasting roles of ICE family proteases and interleukin-1beta in apoptosis induced by trophic factor withdrawal and by copper/zinc superoxide dismutase down-regulation

1 reference

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30 September 2017

On the involvement of a cyclosporin A sensitive mitochondrial pore in myocardial reperfusion injury

1 reference

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30 September 2017

Contribution of the mitochondrial permeability transition to lethal injury after exposure of hepatocytes to t-butylhydroperoxide

1 reference

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30 September 2017

Temporal analysis of events associated with programmed cell death (apoptosis) of sympathetic neurons deprived of nerve growth factor

1 reference

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30 September 2017

Apoptosis: A Basic Biological Phenomenon with Wideranging Implications in Tissue Kinetics

1 reference

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cAMP analogs promote survival and neurite outgrowth in cultures of rat sympathetic and sensory neurons independently of nerve growth factor

1 reference

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30 September 2017

Inhibitors of protein synthesis and RNA synthesis prevent neuronal death caused by nerve growth factor deprivation

1 reference

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30 September 2017

The Ca2+-induced membrane transition in mitochondria. I. The protective mechanisms

1 reference

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The coordinate release of cytochrome c during apoptosis is rapid, complete and kinetically invariant

1 reference

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29 June 2018

The release of cytochrome c from mitochondria during apoptosis of NGF-deprived sympathetic neurons is a reversible event

1 reference

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29 June 2018

Blocking cytochrome c activity within intact neurons inhibits apoptosis.

1 reference

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29 June 2018

Cyclosporin A binding to mitochondrial cyclophilin inhibits the permeability transition pore and protects hearts from ischaemia/reperfusion injury

1 reference

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Attenuation of transient focal cerebral ischemic injury in transgenic mice expressing a mutant ICE inhibitory protein

1 reference

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29 June 2018

Characterization of apoptosis in cultured rat sympathetic neurons after nerve growth factor withdrawal

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2173417

29 June 2018

Evidence for the presence of a reversible Ca2+-dependent pore activated by oxidative stress in heart mitochondria

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2173417

29 June 2018

Inhibition of the c-Jun N-terminal kinase signaling pathway by the mixed lineage kinase inhibitor CEP-1347 (KT7515) preserves metabolism and growth of trophic factor-deprived neurons.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2173417

22 September 2018

Programmed cell death of developing mammalian neurons after genetic deletion of caspases.

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2173417

22 September 2018

Mitochondria are selectively eliminated from eukaryotic cells after blockade of caspases during apoptosis

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2173417

22 September 2018

Cyclosporin A and its nonimmunosuppressive analogue N-Me-Val-4-cyclosporin A mitigate glucose/oxygen deprivation-induced damage to rat cultured hippocampal neurons

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2173417

22 September 2018

Caspase-dependent and -independent death of camptothecin-treated embryonic cortical neurons

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22 September 2018

FK-506, a novel immunosuppressant isolated from a Streptomyces. II. Immunosuppressive effect of FK-506 in vitro

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https://pubmed.ncbi.nlm.nih.gov/12021257

12 December 2020

based on heuristic

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Chronic depolarization prevents programmed death of sympathetic neurons in vitro but does not support growth: requirement for Ca2+ influx but not Trk activation

1 reference

https://pubmed.ncbi.nlm.nih.gov/12021257

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Attenuation of ischemia/reperfusion injury in rats by a caspase inhibitor

1 reference

https://pubmed.ncbi.nlm.nih.gov/12021257

12 December 2020

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inferred from PubMed ID database lookup

Superoxide in apoptosis. Mitochondrial generation triggered by cytochrome c loss

1 reference

https://pubmed.ncbi.nlm.nih.gov/12021257

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inferred from PubMed ID database lookup

Proteases to die for

1 reference

https://pubmed.ncbi.nlm.nih.gov/12021257

12 December 2020

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inferred from PubMed ID database lookup

Evidence of a novel event during neuronal death: development of competence-to-die in response to cytoplasmic cytochrome c

1 reference

https://pubmed.ncbi.nlm.nih.gov/12021257

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

BAX translocation is a critical event in neuronal apoptosis: regulation by neuroprotectants, BCL-2, and caspases

1 reference

https://pubmed.ncbi.nlm.nih.gov/12021257

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1083/JCB.200112130

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

PMC publication ID

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

PubMed publication ID

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

ResearchGate publication ID

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