C9orf72 ablation causes immune dysregulation characterized by leukocyte expansion, autoantibody production, and glomerulonephropathy in mice. (Q36691051)

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scientific article published on 16 March 2016

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English	C9orf72 ablation causes immune dysregulation characterized by leukocyte expansion, autoantibody production, and glomerulonephropathy in mice.	scientific article published on 16 March 2016

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11 March 2020

C9orf72 ablation causes immune dysregulation characterized by leukocyte expansion, autoantibody production, and glomerulonephropathy in mice (English)

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11 March 2020

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Andrew J Murphy

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Amanda Atanasio

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Vilma Decman

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Derek White

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Meg Ramos

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Hoi-Ching Lee

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Chia-Jen Siao

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Susannah Brydges

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Elizabeth LaRosa

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Yu Bai

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Wen Fury

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Patricia Burfeind

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Ralica Zamfirova

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Gregg Warshaw

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Jamie Orengo

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Adelekan Oyejide

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Michael Fralish

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Wojtek Auerbach

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William Poueymirou

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Jan Freudenberg

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Guochun Gong

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Brian Zambrowicz

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George Yancopoulos

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Gavin Thurston

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16 March 2016

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GGGGCC repeat expansion in C9orf72 compromises nucleocytoplasmic transport

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30 September 2017

The C9orf72 repeat expansion disrupts nucleocytoplasmic transport.

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30 September 2017

Isoform-specific antibodies reveal distinct subcellular localizations of C9orf72 in amyotrophic lateral sclerosis

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30 September 2017

C9orf72 ablation in mice does not cause motor neuron degeneration or motor deficits

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Rare Variants of ATG5 Are Likely to Be Associated With Chinese Patients With Systemic Lupus Erythematosus

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Neuropsychiatric lupus: a mosaic of clinical presentations

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Drosha inclusions are new components of dipeptide-repeat protein aggregates in FTLD-TDP and ALS C9orf72 expansion cases

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Aberrant RNA homeostasis in amyotrophic lateral sclerosis: potential for new therapeutic targets?

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C9orf72 nucleotide repeat structures initiate molecular cascades of disease.

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Reduced C9orf72 protein levels in frontal cortex of amyotrophic lateral sclerosis and frontotemporal degeneration brain with the C9ORF72 hexanucleotide repeat expansion

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C9ORF72, implicated in amytrophic lateral sclerosis and frontotemporal dementia, regulates endosomal trafficking

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Deletion of C9ORF72 results in motor neuron degeneration and stress sensitivity in C. elegans

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The mouse C9ORF72 ortholog is enriched in neurons known to degenerate in ALS and FTD.

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The disease-associated r(GGGGCC)n repeat from the C9orf72 gene forms tract length-dependent uni- and multimolecular RNA G-quadruplex structures

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Unconventional translation of C9ORF72 GGGGCC expansion generates insoluble polypeptides specific to c9FTD/ALS

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The C9orf72 GGGGCC repeat is translated into aggregating dipeptide-repeat proteins in FTLD/ALS.

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Expanded GGGGCC hexanucleotide repeat in noncoding region of C9ORF72 causes chromosome 9p-linked FTD and ALS

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The MRL/lpr mouse strain as a model for neuropsychiatric systemic lupus erythematosus

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5 September 2018

Bidirectional transcripts of the expanded C9orf72 hexanucleotide repeat are translated into aggregating dipeptide repeat proteins

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5 September 2018

Loss of function of C9orf72 causes motor deficits in a zebrafish model of amyotrophic lateral sclerosis.

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5 September 2018

hnRNP A3 binds to GGGGCC repeats and is a constituent of p62-positive/TDP43-negative inclusions in the hippocampus of patients with C9orf72 mutations

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5 September 2018

FTD/ALS families are no longer orphaned: the C9ORF72 story.

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5 September 2018

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Multiple Myeloma

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4793236

20 October 2018

Lupus-Prone Mice Have an Abnormal Response to Thioglycolate and an Impaired Clearance of Apoptotic Cells

1 reference

https://api.crossref.org/works/10.1038%2FSREP23204

21 January 2018

Expansion and high proliferative potential of the macrophage system throughout life time of lupus-prone NZB/W and MRL lpr/lpr mice. Lack of down-regulation of extramedullar macrophage proliferation in the postnatal period

1 reference

https://pubmed.ncbi.nlm.nih.gov/26979938

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1038/SREP23204

1 reference

Europe PubMed Central

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:26979938%20AND%20SRC:MED&resulttype=core&format=json

11 March 2020

1 reference

Europe PubMed Central

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:26979938%20AND%20SRC:MED&resulttype=core&format=json

11 March 2020

1 reference

Europe PubMed Central

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:26979938%20AND%20SRC:MED&resulttype=core&format=json

11 March 2020

ResearchGate publication ID

0 references

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