Amyloid-β Peptides and Tau Protein as Biomarkers in Cerebrospinal and Interstitial Fluid Following Traumatic Brain Injury: A Review of Experimental and Clinical Studies (Q36955963)

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scientific article published on 26 June 2013

Language	Label	Description	Also known as
English	Amyloid-β Peptides and Tau Protein as Biomarkers in Cerebrospinal and Interstitial Fluid Following Traumatic Brain Injury: A Review of Experimental and Clinical Studies	scientific article published on 26 June 2013

Statements

scholarly article

1 reference

Europe PubMed Central

17 August 2017

Amyloid-β Peptides and Tau Protein as Biomarkers in Cerebrospinal and Interstitial Fluid Following Traumatic Brain Injury: A Review of Experimental and Clinical Studies (English)

1 reference

Europe PubMed Central

17 August 2017

0 references

traumatic brain injury

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author name string

Parmenion P Tsitsopoulos

1

1 reference

Europe PubMed Central

17 August 2017

Niklas Marklund

2

1 reference

Europe PubMed Central

17 August 2017

publication date

26 June 2013

1 reference

Europe PubMed Central

17 August 2017

Frontiers in Neurology

1 reference

Europe PubMed Central

17 August 2017

4

1 reference

Europe PubMed Central

17 August 2017

79

1 reference

Europe PubMed Central

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The spectrum of disease in chronic traumatic encephalopathy

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Biomarker positive and negative subjects in the ADNI cohort: clinical characterization

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Reference measurement procedures for Alzheimer's disease cerebrospinal fluid biomarkers: definitions and approaches with focus on amyloid β42.

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A paravascular pathway facilitates CSF flow through the brain parenchyma and the clearance of interstitial solutes, including amyloid β.

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A Model for Mild Traumatic Brain Injury that Induces Limited Transient Memory Impairment and Increased Levels of Axon Related Serum Biomarkers

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Apolipoprotein E4 effects in Alzheimer's disease are mediated by synaptotoxic oligomeric amyloid-β

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Alzheimer culprits: cellular crossroads and interplay.

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29 September 2017

Lithium reduces BACE1 overexpression, β amyloid accumulation, and spatial learning deficits in mice with traumatic brain injury

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CSF-biomarkers in Olympic boxing: diagnosis and effects of repetitive head trauma

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Brain injury biomarkers may improve the predictive power of the IMPACT outcome calculator

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Traumatic brain injury: a risk factor for Alzheimer's disease

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Inhibition of JNK by a peptide inhibitor reduces traumatic brain injury-induced tauopathy in transgenic mice

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29 September 2017

Tau elevations in the brain extracellular space correlate with reduced amyloid-β levels and predict adverse clinical outcomes after severe traumatic brain injury

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Detection of Soluble Amyloid-β Oligomers and Insoluble High-Molecular-Weight Particles in CSF: Development of Methods with Potential for Diagnosis and Therapy Monitoring of Alzheimer's Disease

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Dynamic analysis of amyloid β-protein in behaving mice reveals opposing changes in ISF versus parenchymal Aβ during age-related plaque formation

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The Alzheimer's Disease Neuroimaging Initiative: a review of papers published since its inception.

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Distinct temporal and anatomical distributions of amyloid-β and tau abnormalities following controlled cortical impact in transgenic mice

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PET imaging of brain amyloid in dementia: a review

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29 September 2017

Lithium ameliorates neurodegeneration, suppresses neuroinflammation, and improves behavioral performance in a mouse model of traumatic brain injury

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Protein engineering to stabilize soluble amyloid β-protein aggregates for structural and functional studies

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Controlled cortical impact traumatic brain injury in 3xTg-AD mice causes acute intra-axonal amyloid-β accumulation and independently accelerates the development of tau abnormalities

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Widespread τ and amyloid-β pathology many years after a single traumatic brain injury in humans.

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Detrimental effect of genetic inhibition of B-site APP-cleaving enzyme 1 on functional outcome after controlled cortical impact in young adult mice

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The many faces of tau

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29 September 2017

Animal modelling of traumatic brain injury in preclinical drug development: where do we go from here?

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29 September 2017

Amyloid-β as a modulator of synaptic plasticity

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29 September 2017

New perspectives on amyloid-beta dynamics after acute brain injury: moving between experimental approaches and studies in the human brain

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29 September 2017

Traumatic brain injury reduces soluble extracellular amyloid-β in mice: a methodologically novel combined microdialysis-controlled cortical impact study

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Update on the biomarker core of the Alzheimer's Disease Neuroimaging Initiative subjects

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Intraneuronal beta-amyloid accumulation and synapse pathology in Alzheimer's disease

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Traumatic brain injury and amyloid-β pathology: a link to Alzheimer's disease?

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Changes in dendritic complexity and spine morphology in transgenic mice expressing human wild-type tau.

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The role of apolipoprotein E in Alzheimer's disease

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29 September 2017

Update on protein biomarkers in traumatic brain injury with emphasis on clinical use in adults and pediatrics

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29 September 2017

Dementia in a retired world boxing champion: case report and literature review.

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Chronic Traumatic Encephalopathy in Athletes: Progressive Tauopathy After Repetitive Head Injury

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29 September 2017

A neprilysin polymorphism and amyloid-beta plaques after traumatic brain injury

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Amyloid precursor protein secretases as therapeutic targets for traumatic brain injury.

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Amyloid beta protein and tau in cerebrospinal fluid and plasma as biomarkers for dementia: a review of recent literature

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Classification of traumatic brain injury for targeted therapies

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A lack of amyloid beta plaques despite persistent accumulation of amyloid beta in axons of long-term survivors of traumatic brain injury

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Endocytosis is required for synaptic activity-dependent release of amyloid-beta in vivo

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Oligomeric amyloid-beta peptide disrupts phosphatidylinositol-4,5-bisphosphate metabolism

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Multiple proteins implicated in neurodegenerative diseases accumulate in axons after brain trauma in humans

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Rescuing neurons and glia: is inhibition of apoptosis useful?

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Intracellular amyloid-beta in Alzheimer's disease

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Molecular mechanisms for Alzheimer's disease: implications for neuroimaging and therapeutics

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Experimental models of traumatic brain injury: do we really need to build a better mousetrap?

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Enhanced neurofibrillary tangle formation, cerebral atrophy, and cognitive deficits induced by repetitive mild brain injury in a transgenic tauopathy mouse model

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Polymorphisms in neprilysin gene affect the risk of Alzheimer's disease in Finnish patients

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Long-term accumulation of amyloid-beta, beta-secretase, presenilin-1, and caspase-3 in damaged axons following brain trauma

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29 September 2017

CSF markers for incipient Alzheimer's disease

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29 September 2017

Diffuse degeneration of the cerebral white matter in severe dementia following head injury

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Head injury as a risk factor for Alzheimer's disease: the evidence 10 years on; a partial replication.

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29 September 2017

Constructing an ethical stereotaxy for severe brain injury: balancing risks, benefits and access

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29 September 2017

APP processing and synaptic function

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29 September 2017

Alzheimer's disease: molecular understanding predicts amyloid-based therapeutics

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29 September 2017

The amyloid hypothesis of Alzheimer's disease: progress and problems on the road to therapeutics

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29 September 2017

Caspase-3-mediated cleavage of amyloid precursor protein and formation of amyloid Beta peptide in traumatic axonal injury

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29 September 2017

Tau is essential to beta -amyloid-induced neurotoxicity

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29 September 2017

Naturally secreted oligomers of amyloid beta protein potently inhibit hippocampal long-term potentiation in vivo

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29 September 2017

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29 September 2017

Hyperphosphorylation induces self-assembly of tau into tangles of paired helical filaments/straight filaments

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29 September 2017

BACE1 is the major beta-secretase for generation of Abeta peptides by neurons

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29 September 2017

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29 September 2017

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The tangled biology of tau.

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29 September 2017

Association of microglia with amyloid plaques in brains of APP23 transgenic mice

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29 September 2017

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29 September 2017

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29 September 2017

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29 September 2017

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29 September 2017

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29 September 2017

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29 September 2017

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29 September 2017

The occult aftermath of boxing

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29 September 2017

Post-traumatic Alzheimer's disease: preponderance of a single plaque type

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29 September 2017

The delayed neurobehavioural sequelae of traumatic brain injury

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29 September 2017

Axonal response to traumatic brain injury: reactive axonal change, deafferentation, and neuroplasticity.

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29 September 2017

Isolation and quantification of soluble Alzheimer's beta-peptide from biological fluids

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29 September 2017

Epidemiology of Alzheimer's Disease

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29 September 2017

Differential distribution of neurofibrillary tangles in the cerebral cortex of dementia pugilistica and Alzheimer's disease cases

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29 September 2017

Amyloid-beta dynamics correlate with neurological status in the injured human brain

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27 June 2018

Intracerebral microdialysis in neurosurgical intensive care patients utilising catheters with different molecular cut-off (20 and 100 kD).

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27 June 2018

Tau hyperphosphorylation in apolipoprotein E-deficient and control mice after closed head injury.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

27 June 2018

Emergence of immunoreactivities for phosphorylated tau and amyloid-beta protein in chronic stage of fluid percussion injury in rat brain.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

27 June 2018

Twofold overexpression of human beta-amyloid precursor proteins in transgenic mice does not affect the neuromotor, cognitive, or neurodegenerative sequelae following experimental brain injury.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

27 June 2018

Characterization of diffuse axonal pathology and selective hippocampal damage following inertial brain trauma in the pig.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

27 June 2018

A beta 42 is the predominant form of amyloid beta-protein in the brains of short-term survivors of head injury.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

27 June 2018

Is beta-APP a marker of axonal damage in short-surviving head injury?

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

27 June 2018

Diffuse axonal injury and traumatic coma in the primate.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

27 June 2018

Distribution of tau proteins in the normal human central and peripheral nervous system

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

27 June 2018

Neurofibrillary tangles in dementia pugilistica are ubiquitinated

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

27 June 2018

Rapid appearance of beta-amyloid precursor protein immunoreactivity in damaged axons and reactive glial cells in rat brain following needle stab injury.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

27 June 2018

Multiple mechanisms of extracellular tau spreading in a non-transgenic tauopathy model.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Repetitive mild traumatic brain injury augments tau pathology and glial activation in aged hTau mice.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Intranasal administration of nerve growth factor ameliorate β-amyloid deposition after traumatic brain injury in rats.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Sera from amyotrophic lateral sclerosis patients induce the non-canonical activation of NMDA receptors "in vitro".

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

PKC activator therapeutic for mild traumatic brain injury in mice

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Relationship between injury severity and serum tau protein levels in traumatic brain injured rats.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Simvastatin therapy prevents brain trauma-induced increases in beta-amyloid peptide levels.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Tau proteins in serum predict outcome after severe traumatic brain injury.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Expression of amyloid precursor protein after rat traumatic brain injury.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Monitoring of brain interstitial total tau and beta amyloid proteins by microdialysis in patients with traumatic brain injury

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Association of increased cortical soluble abeta42 levels with diffuse plaques after severe brain injury in humans.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Prediction of Alzheimer's disease using the CSF Abeta42/Abeta40 ratio in patients with mild cognitive impairment.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Fluctuations of CSF amyloid-beta levels: implications for a diagnostic and therapeutic biomarker.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Initial CSF total tau correlates with 1-year outcome in patients with traumatic brain injury.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Neurochemical aftermath of amateur boxing.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Synaptic activity regulates interstitial fluid amyloid-beta levels in vivo.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Caspase inhibition therapy abolishes brain trauma-induced increases in Abeta peptide: implications for clinical outcome

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Cerebral microdialysis methodology--evaluation of 20 kDa and 100 kDa catheters

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Cleaved-tau: a biomarker of neuronal damage after traumatic brain injury.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Alzheimer's pathology in human temporal cortex surgically excised after severe brain injury.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Marked increase of beta-amyloid(1-42) and amyloid precursor protein in ventricular cerebrospinal fluid after severe traumatic brain injury.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Vitamin E reduces amyloidosis and improves cognitive function in Tg2576 mice following repetitive concussive brain injury.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Experimental traumatic brain injury in rats stimulates the expression, production and activity of Alzheimer's disease beta-secretase (BACE-1).

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Amyloid beta 1-42 and tau in cerebrospinal fluid after severe traumatic brain injury.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Alterations in cerebrospinal fluid apolipoprotein E and amyloid beta-protein after traumatic brain injury.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Tau immunohistochemistry in acute brain injury

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Amyloid β accumulation in axons after traumatic brain injury in humans

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

CSF Abeta 42 levels correlate with amyloid-neuropathology in a population-based autopsy study.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Extracellular amyloid formation and associated pathology in neural grafts.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Changes in expression of amyloid precursor protein and interleukin-1beta after experimental traumatic brain injury in rats.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Long-term accumulation of amyloid-beta in axons following brain trauma without persistent upregulation of amyloid precursor protein genes.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Apolipoprotein E4 influences amyloid deposition but not cell loss after traumatic brain injury in a mouse model of Alzheimer's disease.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

C-tau biomarker of neuronal damage in severe brain injured patients: association with elevated intracranial pressure and clinical outcome.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Repetitive mild brain trauma accelerates Abeta deposition, lipid peroxidation, and cognitive impairment in a transgenic mouse model of Alzheimer amyloidosis.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

beta-amyloid (Abeta)42(43), abeta42, abeta40 and apoE immunostaining of plaques in fatal head injury.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Traumatic brain injury elevates the Alzheimer's amyloid peptide A beta 42 in human CSF. A possible role for nerve cell injury.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Plasma and cerebrospinal fluid levels of amyloid beta proteins 1-40 and 1-42 in Alzheimer disease.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Accumulation of amyloid beta and tau and the formation of neurofilament inclusions following diffuse brain injury in the pig.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Traumatic brain injury in young, amyloid-beta peptide overexpressing transgenic mice induces marked ipsilateral hippocampal atrophy and diminished Abeta deposition during aging.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Quantification of axonal damage in traumatic brain injury: affinity purification and characterization of cerebrospinal fluid tau proteins.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Experimental brain injury induces expression of amyloid precursor protein, which may be related to neuronal loss in the hippocampus.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Traumatic brain injury increases beta-amyloid peptide 1-42 in cerebrospinal fluid.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Immunohistochemical characterization of alterations in the distribution of amyloid precursor proteins and beta-amyloid peptide after experimental brain injury in the rat.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Beta APP gene expression is increased in the rat brain after motor neuron axotomy.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Apolipoprotein E epsilon 4 allele is associated with deposition of amyloid beta-protein following head injury.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3693096

4 September 2018

Brain trauma in aged transgenic mice induces regression of established abeta deposits

1 reference

https://pubmed.ncbi.nlm.nih.gov/23805125

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Beta-amyloid precursor protein (beta APP) as a marker for axonal injury after head injury

1 reference

https://pubmed.ncbi.nlm.nih.gov/23805125

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Traumatic axonal injury in the spinal cord evoked by traumatic brain injury

1 reference

https://pubmed.ncbi.nlm.nih.gov/23805125

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Documented head injury in early adulthood and risk of Alzheimer's disease and other dementias

1 reference

https://pubmed.ncbi.nlm.nih.gov/23805125

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Apolipoprotein E epsilon 4 and fatal cerebral amyloid angiopathy associated with dementia pugilistica

1 reference

https://pubmed.ncbi.nlm.nih.gov/23805125

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Head trauma as a risk factor for Alzheimer's disease: a collaborative re-analysis of case-control studies. EURODEM Risk Factors Research Group

1 reference

https://pubmed.ncbi.nlm.nih.gov/23805125

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Biomechanical analysis of experimental diffuse axonal injury

1 reference

https://pubmed.ncbi.nlm.nih.gov/23805125

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Amyloid beta 1-42 and tau in cerebrospinal fluid after severe traumatic brain injury

1 reference

https://pubmed.ncbi.nlm.nih.gov/23805125

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Diffuse axonal injury in head injury: definition, diagnosis and grading

1 reference

https://pubmed.ncbi.nlm.nih.gov/23805125

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Traumatic brain injury in rat produces changes of beta-amyloid precursor protein immunoreactivity

1 reference

https://pubmed.ncbi.nlm.nih.gov/23805125

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.3389/FNEUR.2013.00079

1 reference

Europe PubMed Central

17 August 2017

1 reference

Europe PubMed Central

17 August 2017

1 reference

Europe PubMed Central

17 August 2017

ResearchGate publication ID

0 references

Sitelinks

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