Ca(2+) signaling in striated muscle: the elusive roles of triadin, junctin, and calsequestrin (Q37478186)

20 August 2017

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title

Ca(2+) signaling in striated muscle: the elusive roles of triadin, junctin, and calsequestrin (English)

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20 August 2017

Nicole A Beard

1

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PubMed ID

19434403

20 August 2017

Lan Wei

series ordinal

2

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PubMed ID

19434403

20 August 2017

Angela Fay Dulhunty

series ordinal

3

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20 August 2017

12 May 2009

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European Biophysics Journal

20 August 2017

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20 August 2017

39

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20 August 2017

1

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20 August 2017

27-36

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https://scigraph.springernature.com/pub.10.1007/s00249-009-0449-6

20 August 2017

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cites work

Casq2 deletion causes sarcoplasmic reticulum volume increase, premature Ca2+ release, and catecholaminergic polymorphic ventricular tachycardia

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12 December 2020

Crystal structure of calsequestrin from rabbit skeletal muscle sarcoplasmic reticulum

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12 December 2020

Negatively charged amino acids within the intraluminal loop of ryanodine receptor are involved in the interaction with triadin

1 reference

12 December 2020

Calsequestrin mutant D307H exhibits depressed binding to its protein targets and a depressed response to calcium

1 reference

12 December 2020

Calsequestrin: more than 'only' a luminal Ca2+ buffer inside the sarcoplasmic reticulum

1 reference

12 December 2020

Calsequestrin 2 (CASQ2) mutations increase expression of calreticulin and ryanodine receptors, causing catecholaminergic polymorphic ventricular tachycardia

1 reference

12 December 2020

Molecular cloning of cDNA encoding a 55-kDa multifunctional thyroid hormone binding protein of skeletal muscle sarcoplasmic reticulum

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12 December 2020

The assembly of calcium release units in cardiac muscle

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12 December 2020

Impaired relaxation in transgenic mice overexpressing junctin.

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12 December 2020

Junctin and triadin each activate skeletal ryanodine receptors but junctin alone mediates functional interactions with calsequestrin

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12 December 2020

Cardiac remodeling and atrial fibrillation in transgenic mice overexpressing junctin

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12 December 2020

Overexpression of junctin causes adaptive changes in cardiac myocyte Ca(2+) signaling

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12 December 2020

Unique isoform-specific properties of calsequestrin in the heart and skeletal muscle

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12 December 2020

Biochemical characterization of ultrastructural localization of a major junctional sarcoplasmic reticulum glycoprotein (triadin)

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12 December 2020

Knocking down type 2 but not type 1 calsequestrin reduces calcium sequestration and release in C2C12 skeletal muscle myotubes

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12 December 2020

Characterization study of the ryanodine receptor and of calsequestrin isoforms of mammalian skeletal muscles in relation to fibre types

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12 December 2020

Comparing skeletal and cardiac calsequestrin structures and their calcium binding: a proposed mechanism for coupled calcium binding and protein polymerization

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12 December 2020

Stress and high heart rate provoke ventricular tachycardia in mice expressing triadin

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12 December 2020

Junctin is a prominent regulator of contractility in cardiomyocytes

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12 December 2020

Localization and partial characterization of the oligomeric disulfide-linked molecular weight 95,000 protein (triadin) which binds the ryanodine and dihydropyridine receptors in skeletal muscle triadic vesicles

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12 December 2020

The conformation of calsequestrin determines its ability to regulate skeletal ryanodine receptors

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12 December 2020

Triadin overexpression stimulates excitation-contraction coupling and increases predisposition to cellular arrhythmia in cardiac myocytes

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12 December 2020

Biochemical characterization and molecular cloning of cardiac triadin

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12 December 2020

Luminal Ca2+ regulation of single cardiac ryanodine receptors: insights provided by calsequestrin and its mutants

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12 December 2020

Sarcoplasmic reticulum calcium overloading in junctin deficiency enhances cardiac contractility but increases ventricular automaticity

1 reference

12 December 2020

A mutation in calsequestrin, CASQ2D307H, impairs Sarcoplasmic Reticulum Ca2+ handling and causes complex ventricular arrhythmias in mice

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12 December 2020

Disulfide bonds, N-glycosylation and transmembrane topology of skeletal muscle triadin

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12 December 2020

Altered stored calcium release in skeletal myotubes deficient of triadin and junctin

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12 December 2020

Functional interaction of the cytoplasmic domain of triadin with the skeletal ryanodine receptor

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12 December 2020

A variably spliced region in the type 1 ryanodine receptor may participate in an inter-domain interaction.

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12 December 2020

Effects of anti-triadin antibody on Ca2+ release from sarcoplasmic reticulum.

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12 December 2020

Altered mRNA splicing of the skeletal muscle ryanodine receptor and sarcoplasmic/endoplasmic reticulum Ca2+-ATPase in myotonic dystrophy type 1.

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12 December 2020

Alternative splicing of RyR1 alters the efficacy of skeletal EC coupling.

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12 December 2020

Abnormal calcium signaling and sudden cardiac death associated with mutation of calsequestrin

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12 December 2020

Calsequestrin is an inhibitor of skeletal muscle ryanodine receptor calcium release channels

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12 December 2020

Modulation of SR Ca release by luminal Ca and calsequestrin in cardiac myocytes: effects of CASQ2 mutations linked to sudden cardiac death.

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12 December 2020

Triadin (Trisk 95) overexpression blocks excitation-contraction coupling in rat skeletal myotubes

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12 December 2020

Reorganized stores and impaired calcium handling in skeletal muscle of mice lacking calsequestrin-1

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12 December 2020

Coexistence of two calsequestrin isoforms in rabbit slow-twitch skeletal muscle fibers

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12 December 2020

Complex formation between junctin, triadin, calsequestrin, and the ryanodine receptor. Proteins of the cardiac junctional sarcoplasmic reticulum membrane

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12 December 2020

Characterization of cardiac calsequestrin

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12 December 2020

Characterization of human cardiac calsequestrin and its deleterious mutants

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12 December 2020

Catecholaminergic polymorphic ventricular tachycardia-related mutations R33Q and L167H alter calcium sensitivity of human cardiac calsequestrin

1 reference

12 December 2020

Triadins are not triad-specific proteins: two new skeletal muscle triadins possibly involved in the architecture of sarcoplasmic reticulum

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12 December 2020

Primary structure and topological analysis of a skeletal muscle-specific junctional sarcoplasmic reticulum glycoprotein (triadin).

1 reference

12 December 2020

Muscle-specific GSTM2-2 on the luminal side of the sarcoplasmic reticulum modifies RyR ion channel activity.

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12 December 2020

Depletion "skraps" and dynamic buffering inside the cellular calcium store.

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12 December 2020

Unexpected structural and functional consequences of the R33Q homozygous mutation in cardiac calsequestrin: a complex arrhythmogenic cascade in a knock in mouse model

1 reference

12 December 2020

Calsequestrin binds to monomeric and complexed forms of key calcium-handling proteins in native sarcoplasmic reticulum membranes from rabbit skeletal muscle

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12 December 2020

Triadins modulate intracellular Ca(2+) homeostasis but are not essential for excitation-contraction coupling in skeletal muscle

1 reference

12 December 2020

Cardiac hypertrophy and impaired relaxation in transgenic mice overexpressing triadin 1

1 reference

12 December 2020

Complete amino acid sequence of canine cardiac calsequestrin deduced by cDNA cloning

1 reference

12 December 2020

Regulation of ryanodine receptors by calsequestrin: effect of high luminal Ca2+ and phosphorylation.

1 reference

12 December 2020

The role of calsequestrin, triadin, and junctin in conferring cardiac ryanodine receptor responsiveness to luminal calcium

1 reference

12 December 2020

Calsequestrin and the calcium release channel of skeletal and cardiac muscle

1 reference

12 December 2020

Dual regulation of the skeletal muscle ryanodine receptor by triadin and calsequestrin

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12 December 2020

Triadin binding to the C-terminal luminal loop of the ryanodine receptor is important for skeletal muscle excitation contraction coupling

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12 December 2020

10.1007/S00249-009-0449-6

Identifiers

DOI

1 reference

20 August 2017

1 reference

PubMed ID

19434403