Molecular mechanism of NLRP3 inflammasome activation (Q37768702)

22 August 2017

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title

Molecular mechanism of NLRP3 inflammasome activation (English)

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22 August 2017

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2

Richard A Flavell

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22 August 2017

Chengcheng Jin

1

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22 August 2017

30 June 2010

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Journal of Clinical Immunology

22 August 2017

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22 August 2017

30

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22 August 2017

5

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22 August 2017

628-631

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https://scigraph.springernature.com/pub.10.1007/s10875-010-9440-3

22 August 2017

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Malarial hemozoin is a Nalp3 inflammasome activating danger signal

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Inflammasomes: guardians of cytosolic sanctity

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Human monocyte interleukin-1beta posttranslational processing. Evidence of a volume-regulated response

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The Nalp3 inflammasome is essential for the development of silicosis

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Inflammasome activation. The missing link: how the inflammasome senses oxidative stress

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Crucial role for the Nalp3 inflammasome in the immunostimulatory properties of aluminium adjuvants

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Gout-associated uric acid crystals activate the NALP3 inflammasome

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Silica crystals and aluminum salts activate the NALP3 inflammasome through phagosomal destabilization

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Thioredoxin-interacting protein links oxidative stress to inflammasome activation

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Active caspase-1 is a regulator of unconventional protein secretion

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Disease-associated mutations in CIAS1 induce cathepsin B-dependent rapid cell death of human THP-1 monocytic cells

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Caspase-1 activation of lipid metabolic pathways in response to bacterial pore-forming toxins promotes cell survival

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Innate immune activation through Nalp3 inflammasome sensing of asbestos and silica

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A crucial function of SGT1 and HSP90 in inflammasome activity links mammalian and plant innate immune responses

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Bacterial RNA and small antiviral compounds activate caspase-1 through cryopyrin/Nalp3

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Pyroptosis: host cell death and inflammation

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Cryopyrin activates the inflammasome in response to toxins and ATP

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CA-074Me protection against anthrax lethal toxin

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Activation of the NALP3 inflammasome is triggered by low intracellular potassium concentration

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Differing caspase-1 activation states in monocyte versus macrophage models of IL-1beta processing and release

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Critical role for NALP3/CIAS1/Cryopyrin in innate and adaptive immunity through its regulation of caspase-1

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Critical role for Cryopyrin/Nalp3 in activation of caspase-1 in response to viral infection and double-stranded RNA

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Activation of the IL-1β-Processing Inflammasome Is Involved in Contact Hypersensitivity

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Superoxide dismutase 1 regulates caspase-1 and endotoxic shock

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The inflammasome recognizes cytosolic microbial and host DNA and triggers an innate immune response

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Critical role for cathepsin B in mediating caspase-1-dependent interleukin-18 maturation and caspase-1-independent necrosis triggered by the microbial toxin nigericin

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Molecular mechanisms involved in inflammasome activation

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Human monocyte stimulus-coupled IL-1beta posttranslational processing: modulation via monovalent cations

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ATP activates a reactive oxygen species-dependent oxidative stress response and secretion of proinflammatory cytokines in macrophages

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The inflammasome mediates UVB-induced activation and secretion of interleukin-1beta by keratinocytes

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The NALP3 inflammasome is involved in the innate immune response to amyloid-beta

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12 December 2020

NLRP3/cryopyrin is necessary for interleukin-1beta (IL-1beta) release in response to hyaluronan, an endogenous trigger of inflammation in response to injury

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Syk kinase signalling couples to the Nlrp3 inflammasome for anti-fungal host defence

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Identifying infection-associated genes of Candida albicans in the postgenomic era.

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12 December 2020

10.1007/S10875-010-9440-3

Identifiers

DOI

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22 August 2017

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PubMed ID

20589420