Bicarbonate therapy for prevention of chronic kidney disease progression (Q38150972)

29 August 2017

title

Bicarbonate therapy for prevention of chronic kidney disease progression (English)

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chronic renal insufficiency

29 August 2017

main subject

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1

Igor Łoniewski

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29 August 2017

Donald E Wesson

2

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29 August 2017

language of work or name

English

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publication date

9 October 2013

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29 August 2017

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29 August 2017

volume

85

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29 August 2017

issue

3

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29 August 2017

page(s)

529-535

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https://scigraph.springernature.com/pub.10.1038/ki.2013.401

29 August 2017

exact match

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cites work

Metabolic acidosis and kidney disease: does bicarbonate therapy slow the progression of CKD?

1 reference

https://api.crossref.org/works/10.1038%2FKI.2013.401

7 January 2021

Dietary protein causes a decline in the glomerular filtration rate of the remnant kidney mediated by metabolic acidosis and endothelin receptors.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2013.401

7 January 2021

Amelioration of metabolic acidosis in patients with low GFR reduced kidney endothelin production and kidney injury, and better preserved GFR.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2013.401

7 January 2021

Association of serum bicarbonate levels with mortality in patients with non-dialysis-dependent CKD.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2013.401

7 January 2021

Serum bicarbonate and mortality in stage 3 and stage 4 chronic kidney disease

1 reference

https://api.crossref.org/works/10.1038%2FKI.2013.401

7 January 2021

High serum bicarbonate level within the normal range prevents the progression of chronic kidney disease in elderly chronic kidney disease patients

1 reference

https://api.crossref.org/works/10.1038%2FKI.2013.401

7 January 2021

Association of serum bicarbonate with risk of renal and cardiovascular outcomes in CKD: a report from the Chronic Renal Insufficiency Cohort (CRIC) study.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2013.401

7 January 2021

Net endogenous acid production is associated with a faster decline in GFR in African Americans

1 reference

https://api.crossref.org/works/10.1038%2FKI.2013.401

7 January 2021

Acid retention accompanies reduced GFR in humans and increases plasma levels of endothelin and aldosterone

1 reference

https://api.crossref.org/works/10.1038%2FKI.2013.401

7 January 2021

Increased tissue acid mediates a progressive decline in the glomerular filtration rate of animals with reduced nephron mass.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2013.401

7 January 2021

Angiotensin II receptors mediate increased distal nephron acidification caused by acid retention

1 reference

https://api.crossref.org/works/10.1038%2FKI.2013.401

7 January 2021

Pathophysiology of chronic tubulo-interstitial disease in rats. Interactions of dietary acid load, ammonia, and complement component C3.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2013.401

7 January 2021

Bicarbonate supplementation slows progression of CKD and improves nutritional status

1 reference

https://api.crossref.org/works/10.1038%2FKI.2013.401

7 January 2021

Daily oral sodium bicarbonate preserves glomerular filtration rate by slowing its decline in early hypertensive nephropathy.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2013.401

7 January 2021

Dietary acid reduction with fruits and vegetables or bicarbonate attenuates kidney injury in patients with a moderately reduced glomerular filtration rate due to hypertensive nephropathy

1 reference

https://api.crossref.org/works/10.1038%2FKI.2013.401

7 January 2021

Effects of oral sodium bicarbonate in patients with CKD

1 reference

https://api.crossref.org/works/10.1038%2FKI.2013.401

7 January 2021

A comparison of treating metabolic acidosis in CKD stage 4 hypertensive kidney disease with fruits and vegetables or sodium bicarbonate

1 reference

https://api.crossref.org/works/10.1038%2FKI.2013.401

7 January 2021

Short- and long-term effects of alkali therapy in chronic kidney disease: a systematic review

1 reference

https://api.crossref.org/works/10.1038%2FKI.2013.401

7 January 2021

Deletion of the pH sensor GPR4 decreases renal acid excretion

1 reference

https://api.crossref.org/works/10.1038%2FKI.2013.401

7 January 2021

Metabolic alkalosis due to absorption of "nonabsorbable" antacids

1 reference

https://api.crossref.org/works/10.1038%2FKI.2013.401

7 January 2021

Hypercalcaemia and metabolic alkalosis with betel nut chewing: emphasis on its integrative pathophysiology

1 reference

https://api.crossref.org/works/10.1038%2FKI.2013.401

7 January 2021

Hyponatremia, hypernatremia, and mortality in patients with chronic kidney disease with and without congestive heart failure

1 reference

https://api.crossref.org/works/10.1038%2FKI.2013.401

7 January 2021

Consequences and therapy of the metabolic acidosis of chronic kidney disease

1 reference

https://api.crossref.org/works/10.1038%2FKI.2013.401

7 January 2021

Influence of steady-state alterations in acid-base equilibrium on the fate of administered bicarbonate in the dog.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2013.401

7 January 2021

Observational studies versus randomized controlled trials: avenues to causal inference in nephrology

1 reference

https://api.crossref.org/works/10.1038%2FKI.2013.401

7 January 2021

Identifiers

DOI

10.1038/KI.2013.401

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Dimensions Publication ID

29 August 2017

1004459086

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1 reference